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Spiking neurons. Oscilloscope readings taken off the analog circuits of the SlugBug's brain look amazingly biologically. Experiments: Iguana Robotics donated 7 SlugBugs see figure 1 ; to the workshop. Students were taught to program neuronal networks to create network oscillators or Central Pattern Generators. The SlugBug brain was also interfaced to a 4 legged robot called lobstrosity to create a novel walking platform. Students also mounted whiskers on SlugBug and successfully modulated the behavior of the robot using sensory input that modulated an onboard CPG circuit. Using SlugBug, students had a chance to explore the connection of basic membrane biophysics to behavior.
26. Menard, J., D. J. Campbell, M. Azizi, and M. F. Gonzales. Synergistic effects of ACE inhibition and Ang II antagonism on blood pressure, cardiac weight, and renin in spontaneously hypertensive rats. Circulation 96: 30723078, 1997. Milavetz, J. J., T. E. Raya, C. S. Johnson, E. Morkin, and S. Goldman. Survival after myocardial infarction in rats: captopril versus losartan. J. Am. Coll. Cardiol. 27: 714719, 1996. Obayashi, M., M. Yano, M. Kohno, S. Kobayashi, T. Tanigawa, K. Hironaka, T. Ryouke, and M. Matsuzaki. Dosedependent effect of ANG II-receptor antagonist on myocyte remodeling in rat cardiac hypertrophy. Am. J. Physiol. 273 Heart Circ. Physiol. 42 ; : H1824H1831, 1997. 29. Pitt, B., R. Segal, F. A. Martinez, G. Meurers, A. J. Cowley, I. Thomas, P. C. Deedwania, D. E. Ney, D. B. Snavely, and P. I. Chang. Randomised trial of losartan versus captopril in patients over 65 with heart failure Evaluation of Losartan in the Elderly Study, ELITE ; . Lancet 349: 747752, 1997. Regitz-Zagrosek, V., N. Freidel, A. Heymann, P. Bauer, M. Neub, A. Rolfs, C. Steffen, A. Hildebrandt, R. Hetzer, and E. Fleck. Regulation, chamber localization, and subtype distribution of angiotensin II receptors in human hearts. Circulation 91: 14611471, 1995. Sabbah, H. N., V. G. Sharov, M. Lesch, and S. Goldstein. Progression of heart failure: a role for interstitial fibrosis. Mol. Cell. Biochem. 147: 2934, 1995. Sabbah, H. N., H. Shimoyama, T. Kono, R. C. Gupta, V. G. Sharov, G. Scicli, T. B. Levine, and S. Goldstein. Effects of long-term monotherapy with enalapril, metoprolol, and digoxin on the progression of left ventricular dysfunction and dilation in dogs with reduced ejection fraction. Circulation 89: 28522859, 1994. Sabbah, H. N., P. D. Stein, T. Kono, M. Gheorghiade, T. B. Levine, S. Jafri, E. T. Hawkins, and S. Goldstein. A canine model of chronic heart failure produced by multiple sequential coronary microembolizations. Am. J. Physiol. 260 Heart Circ. Physiol. 29 ; : H1379H1384, 1991. 34. Sechi L. A., C. A. Griggin, E. F. Grady, J. E. Kalinyak, and M. Schambelan. Characterization of angiotensin II receptor subtypes in rat heart. Circ. Res. 71: 14821489, 1992.
On Oct. 12, the Board of Directors created a new officer position at Kanuga, vice president for camping and outdoor education, and unanimously approved Paul Bockoven for the new post. "The board's action was taken in recognition of the broadening Bockoven scope of Kanuga's outdoor activities and the need to provide senior-level leadership for those important ministries, " said Stan Hubbard, president. Camp Kanuga, Camp Bob and the Trailblazer Adventure have roots reaching back to Kanuga's founding in 1928 and are a primary way that Kanuga reaches a diverse population of children and youth. Residential environmental and science education came later but also are central to Kanuga's purpose. Kanuga is stepping up its environmental stewardship efforts. "Caring for this beautiful place and educating others about stewardship of God's creation are central to our purpose, " Hubbard explained. "We have begun partnering with a variety of specialists to improve our 1, 400 acres of private forest, increase our use of sustainable energy, conserve our buildings and reduce waste. This fall we initiated a partnership aimed at increasing the use of local fruits and vegetables in all of our kitchens. Our goal is to serve healthier food while reducing use of fossil fuel to import produce from distant processing plants." "Paul is the natural choice to lead this important cluster of ministries, " Hubbard continued. "He has shown an extraordinary mix of vision, business acumen, leadership and passion in his work at Kanuga." With 14 years on Kanuga staff, Paul has certifications in adventure-based education, rock-climbing instruction, emergency medical response and rescue, as well as membership in the American Camp Association and the Association for Experiential Education. He previously served on the board of Hendersonville's Environmental and Conservation Organization. Paul met his wife, Beth, at Kanuga. They live here yearround with their daughters, Carson, age 8, and Cecilia, 6.
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Possible mechanisms underlying these observations will be presented. Acidosis: Effect on Passive Pressure-Volume Relationships Both IA and RA rotated left ventricular end-diastolic passive ; pressure-volume relationships leftward Table 3 and Fig. 3 ; , indicating an increase in passive chamber stiffness. Because coronary perfusion pressure Pcor ; was constant, the decrease in Pv with acidosis has a potential to increase myocardial volume intravascular, extravascular, or both ; , which can augment both systolic and diastolic Pv at a fixed Vv 22 ; . previous study, we showed that, at a given Vv, Pmax reductions of 3540%, induced with hypocalcemia 0.63 mM ; , lead to 1825% increases in Ped [Ca2 ] and 2530% increases in Ep 43 ; Similar increases in Ped and Ep were found when Pcor was increased 4 ; . In the present study, RA caused a 23% reduction in Pmax coincident with a 40% increase in Ped and a 28% increase in Ep. Similarly, IA caused Pmax to fall 26% while Ped and Ep increased 25 and 28%, respectively. Thus changing the pressure gradient between Pcor and Pv in this study was accompanied by changes in diastolic properties similar to those seen previously 43 ; .1 Furthermore, acidosis does not appear to alter passive force at a fixed muscle length in isolated cardiac muscle studies [e.g., see Fig. 1 in Ricciardi et al. 39 ; and Fig. 1 in Orchard and Kentish 34 ; ], indicating no direct effects of acidosis on passive muscle properties. These considerations lead us to suggest that changes in left ventricular passive behavior in the present study are secondary to myocardial turgor; it is not necessary to invoke any direct effects of pH. That LA showed little changes in Ped or Ep is probably due to the significantly smaller decrease in Pmax. Respiratory and Inorganic Acidosis: Effect on Active Pressure Development and Relaxation All data were collected during steady-state conditions, at least 12 min after a change of perfusate. This is important in the light of observations made by Orchard 33 ; in an isolated cardiac muscle preparation. A rather long transient response was observed in both intracellular free Ca2 signal and developed tension after exposure to respiratory acidosis. In the eventual steady state, the free Ca2 signal was slightly elevated and prolonged compared with that of the control, and tension was reduced. Similar behavior was observed with inorganic and lactic acidosis as well. If this prolongation was due to impaired Ca2 uptake by the sarcoplasmic reticulum SR ; , the relaxation should.
Since this paper was written it has been shown Unit a rise of nervous pressure causes cardiac acceleration even in the presence of the vagus J. J. Jones, Ph.D. thesis, London, 1957.
S-Thalidomide has proven efficacy in multiple myeloma. Although it has both antiangiogenic and pro-apoptotic effects, its primary mode of therapeutic action remains unclear. We have investigated the changes to the expression of genes involved with these cellular processes following culture with s-thalidomide in the U266 MM cell line. Cells were cultured with sthalidomide 01000 mM ; , and cell parameters, including apoptosis, were assessed on day 3. RNA was extracted from cells cultured for 24 h at the IC50 concentration of s-thalidomide, and changes to gene expression were investigated by microarray methodologies. A reduction in cell viability was observed in U266 cells cultured with s-thalidomide IC50: 362 mM ; , which were mirrored by significant increases in apoptosis for example, 200 mM on day 3: 40.373.1% vs. 3.270.4% on day 0; Po0.001 ; . There were changes in the expression profile of genes involved in angiogenesis and apoptosis, but the changes were most dramatic in the apoptotic genes. In particular, the expression of I-kB kinase was decreased by two-fold, which was associated with a four-fold decrease in NF-kB expression. These data correlated with immunoblotting analyses, which showed significant increases in I-kB protein levels and decreased NF-kB activity. Additionally, the Bax : Bcl-2 ratio was significantly increased. Our data suggest that both angiogenic and apoptotic genes and proteins are affected by s-thalidomide. Additionally, a dramatic decrease in Bcl-2 expression with s-thalidomide suggests a possible enhancement of cytotoxic effect if combined with other cytotoxic agents. The Hematology Journal 2004 ; 5, 247254. doi: 10.1038 sj.thj.6200351 Keywords: s-Thalidomide; multiple myeloma; microarray methodologies; NF-kB and amikacin.
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Is needed to join a disability evaluation committee. This is a 9 type position five days a week, and, involves no emergency or surgical type work. It is an ideal position for someone who wants to slow down for reasons of age or health, or, who is just plain tired of running. Please write: David Jacknow, Chairman Disability Detroit Evaluation Industrial Committee Clinic PC M.D.
Psoriasis. J Health Syst Pharm 2000; 57: 645659. Greaves MW, Weinstein GD. Treatment of psoriasis. N Engl J Med 1995; 332: 581588. National Psoriasis Foundation, 2003. Available at: psoriasis resources statistics. Accessed March 29, 2003. 4. Austin LM, Ozawa M, Kikuchi T, et al. The majority of epidermal T cells in psoriasis vulgaris lesions can produce type 1 cytokines, interferon-gamma, interleukin-2, and tumor necrosis factoralpha, defining TC1 cytotoxic T lymphocyte ; and TH1 effector populations: A type 1 differentiation bias is also measured in circulating blood T cells in psoriatic patients. J Invest Dermatol 1999; 113: 752759. Friedrich M, Krammig S, Henze M, et al. Flow cytometric characterization of lesional T cells in psoriasis: Intracellular cytokine and surface antigen expression indicates an activated, memory effector type 1 immunophenotype. Arch Dermatol Res 2000; 292: 519521. Stern RS, Liebman EJ, Vakeva L. Oral psoralen and ultraviolet-A light PUVA ; treatment of psoriasis and persistent risk of non-melanoma skin cancer. PUVA Follow-up Study. J Natl Cancer Inst 1998; 90: 12781284. Van Dooren-Greebe RJ, Kuijpers AL, Mulder J, et al. Methotrexate revisited: Effects of long-term treatment in psoriasis. Br J Dermatol 1994; 130: 204210. Lebwohl M, Ellis C, Gottlieb A, Koo J, et al. Cyclosporine Consensus Conference: With emphasis on the treatment of psoriasis. J Acad Dermatol 1998; 39: 464475. Dutz JP, Ho VC. Immunosuppressive agents in dermatology: An update. Dermatol Clin 1998; 16: 235251. Amevive alefacept ; package insert. Cambridge, MA: Biogen, Inc.; February 2003. 11. Miller GT, Hochman PS, Meier W, et al. Specific interaction of lymphocyte functionassociated antigen 3 with CD2 can inhibit T-cell responses. J Exp Med 1993; 178: 211222. Gordon K, Vaishnaw A, McCormick T, et al. Alefacept selectively reduces memory-effector CD45RO + ; T cells: A mechanism for improving clinical symptoms of psoriasis without impairing immune function. Poster P574. Presented at the 60th annual meeting of the American Academy of Dermatology, February 2227, 2002, New Orleans, LA. da Silva AJ, Brickelmaier M, Majeau GR, et al. Alefacept, an immunomodulatory recombinant LFA-3 IgG1 fusion protein, induces CD12 signaling and CD2 CD12-dependent apoptosis of CD2 + cells. J Immunol 2002; 168: 44624471. Ellis CN, Krueger GG. Treatment of chronic plaque psoriasis by selective targeting of memory effector T lymphocytes. N Engl J Med 2001; 345: 248255. Lowe N, Griffiths CEM, Gottlieb AB. Alefacept human LFA-3 IgG1 ; produces selective reductions in memory-effector CD45RO + ; T cells that correlate with clinical improvement in psoriasis. Poster P824. Presented at the 63rd annual meeting of the Society of Investigative Dermatology, May 1518, 2002, Los Angeles, CA. Krueger JG, Gilleaudeau P, Kikuchi T. Psoriasis-related subpopulations of memory CD4 and CD8 T cells are selectively reduced by alefacept. Poster P823. Presented at the 63rd annual meeting of the Society of Investigative Dermatology, May 1518, 2002, Los Angeles, CA. Ellis CN, Mordin MM, Adler EY. Effects of alefacept on health-related quality of life in patients with psoriasis: Results from a randomized, placebo-controlled phase II trial. J Clin Dermatol 2003; 4: 131139. Krueger GG, Papp KA, Stough DB, et al. A randomized, double-blind, placebocontrolled phase III study evaluating efficacy and tolerability of two courses of alefacept in patients with chronic plaque psoriasis. J Acad Dermatol 2002; 47: 821833. Lowe N, Krueger GG, Menter A, et al. Results of interim analyses of repeat courses of intramuscular alefacept therapy for the treatment of chronic plaque psoriasis. Poster P579. Presented at the 60th annual meeting of the American Academy of Dermatology, February 2227, 2002, New Orleans, LA. Goedkoop AY, de Rie MA, Picavet DI, et al. Alefacept treatment in psoriatic arthritis: Clinical improvement of skin lesions and arthritis correlates with reduction in CD45RO + T cells in epidermis and synovial tissue. Poster P593. Presented at the 61 st annual meeting of the American Academy of Dermatology, March 2126, 2003, San Francisco, CA and aminoglutethimide.
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Peripheral vascular disease and patients being evaluated for major surgical procedures in order to reduce complications during anesthesia and post-operative recovery.
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Meta-analysis of biologic treatments for psoriasis With many biological therapies now available for the treatment of moderate to severe psoriasis and little head-to-head data, many physicians are trying to sort out existing clinical information to weigh and compare relative safety and efficacy levels among these treatments. Treatments of interest included J&J Schering-Plough's Remicade infliximab ; , Abbott CAT's Humira adalimumab ; , Amgen Wyeth's Enbrel etanercept ; , Genentech Merck Serono's Raptiva efalizumab ; , and Astellas' Amevive alefacept ; . While there is no conclusive evidence to support the superior efficacy of one treatment over the other, Mark Bechtel, M.D., Director of Dermatology at The Ohio State University College of Medicine spoke on a meta-analysis of existing clinical information to compare the efficacy of these treatments in severe psoriasis. It was highlighted that several of the studies may have varied patient demographics, time frames for evaluation of efficacy endpoints, and onset of action which make them difficult to compare across the board. However, as all patients were objectively scored they were able to compare the percentage of patients achieving PASI Psoriasis Area and Severity Index ; 75 across clinical studies at two time-points 10-12 weeks and 24-26 weeks.
AMBISOME 50 MG VIAL, 9 amcinonide 0.1% crm lot oint, 35 AMEVIVE 15 MG VIAL, 15 amigesic, 48 amikacin 50mg ml vial, 7 amiloride hcl 5 mg tablet, 31 amiloride hcl hctz 5 50 tab, 31 aminate w 90 mg iron tab sa, 54 AMINESS 5.2% IV SOLUTION, 48 aminophylline, 59 AMINOSYN II IN D5W IV, 48 AMINOSYN II IV SOLUTION, 48 AMINOSYN IV SOLUTION, 48 AMINOSYN M 3.5% IV SOLUTION, 48 AMINOSYN-HBC 7% IV SOLUTION, 48 AMINOSYN-HF 8% IV SOLUTION, 48 AMINOSYN-PF 10% IV SOLUTION, 48 AMINOSYN-PF 7% IV SOLUTION, 48 AMINOSYN-RF IV SOLUTION, 48 amiodarone, 28 AMITIZA, 42 amitrip cdp 12.5-5 tablet, 22 amitrip cdp 25-10 tablet, 22 amitrip perphen, 22 amitrip-cdp 25-10 tablet, 22 amitriptyline, 23 ammonium lactate crm & lot, 36 AMNESTEEM, 35 amoclan 200-28.5 5 suspension, 12 amoclan 400-57 5 suspension, 12 amox tr-k clv, 12 amoxapine, 23 amoxicillin, 12 AMOXIL, 12 amphetamine salts, 27 AMPHOTEC, 9 amphotericin B 50mg vial, 9 ampicillin, 12 Page 62 of 83 and amoxapine.
Sinha-Hikim et al: Testosterone Induces Muscle Fiber Hypertrophy INTRODUCTION Testosterone administration in replacement doses to hypogonadal men 11-12, 26, 43, ; , and in supraphysiologic doses to healthy, eugonadal men 10, 16, 18, ; is associated with significant increases in fat-free mass and muscle size. Similarly, testosterone replacement in men infected with the human immunodeficiency virus, who are experiencing weight loss and have low testosterone levels, induces significant gains in lean body mass with concomitant increases in muscle volume 8-9, 19 ; . We have recently demonstrated that changes in circulating testosterone concentrations, induced by combined administration of GnRH agonist and graded doses of testosterone, are associated with testosterone dose- and concentration-dependent changes in fat-free mass and muscle size 7 ; . However, the mechanisms by which testosterone increases muscle mass are not well understood. We do not know whether testosterone-induced increase in muscle mass is due to muscle fiber hypertrophy, muscle cell hyperplasia, or both. The effects of testosterone administration on muscle fiber size and composition in humans are unknown, and the data from experimental animals are both limited and somewhat contradictory. For instance, in a study by Tucek et al. 45 ; , testosterone treatment of castrated rats led to a 15% increase in the weight of the levator ani muscle; this increase in muscle mass was accompanied by an increase in the size of muscle fibers. Tobin et al., 44 ; reported a significant gender difference both in the number of fibers and their average cross-sectional areas for levator ani muscle, and speculated that the higher fiber number and size in the male rats might be related to the higher testosterone levels. However, a recent study did not find any differences in the crosssectional areas of fast and slow-twitch fibers between older men and women with vastly different androgen concentrations 17 ; . Therefore, the overall objective of the present study was to determine whether testosterone-induced increase in muscle size is due to increase in muscle fiber area or number, or 3.
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Weight kg ; BMI kg m2 ; Body fat kg ; Lean body mass kg ; DBP mmHg ; SBP mmHg ; S- total cholesterol mmol l ; S-LDL-cholesterol mmol l ; S-HDL-cholesterol mmol l ; S-Triglycerides mmol l ; S-insulin mU l ; Placebo vs. orlistat weight loss at 12 months 7.2kg vs. 13.0kg ; was not significant and the results of orlistat and placebo were pooled and amprenavir.
Heidelberg. Phone 49-6221-568211, Fax 49-6221-565348. E-mail alexandra kunze med -heidelberg Website space. stroke-trial Location: Europe Number of Centers: 30 Sponsors: BMBF German Ministry of Science ; , DFG German Research Council ; , Guidant, Boston Scientific Dates of Study: 2000 2004.
P 0.027 ; and 4 wk P 0.025 ; rats. All other parameters were the same comparing NT to 1 and 4 wk HT rats. The relationship between the P2, the gain coefficient, and MAP is illustrated in Fig. 2D. There were no differences in any parameters comparing 1 wk to rats. Heart rate. MAP was greater in both HT groups compared with NT rats P 0.001 ; . NT rats had a lower MAP 118 mmHg, SD 9, n 10 ; than 1 wk HT 131 mmHg, SD 4, n 5; P 0.017 ; and 4 wk HT 141 mmHg, SD 11, n 8; P 0.001 ; rats. There were no differences in resting HRs NT 401 bpm, SD 32; 1 wk HT 399 bpm, SD 36; 4 wk HT 397 bpm, SD 34; P 0.969 ; . Table 3 contains the averaged curve fit parameters for baroreflex regulation of HR in anesthetized rats. Curves drawn using the averaged parameters are illustrated in Fig. 3A. There was a significant shift in P3 comparing NT to both 1 wk P 0.049 ; and 4 wk P 0.009 ; HT rats. In contrast to the RSNA curves, P2 was significantly greater in NT compared with 1 wk P 0.007 ; and 4 wk P 0.002 ; HT rats. In addition, the Gmax of the relationship was greater in NT compared with 1 wk P 0.009 ; and 4 wk P 0.001 ; HT rats. There were no differences in any parameters comparing 1 wk to rats. The relationship between the P2, the gain coefficient, and MAP is illustrated in Fig. 3B. Conscious rat studies. The range of HR, the P1 value, was small in the anesthetized, paralyzed rat 22 ; . Therefore, curves relating MAP and HR were generated in conscious NT rats n 5 ; and HT rats after 1 wk n and 4 wk n hypertension. MAP was greater in both HT groups compared with NT rats NT 102 mmHg, SD 6; 1 wk HT 131 mmHg, SD 10; 4 wk HT 151 mmHg, SD 22; P 0.001 ; , and there was no difference in resting HR NT 362 bpm, SD 13; 1 wk HT 352 bpm, SD 36; 4 wk HT 366 bpm, SD 31; P 0.750 ; . Fig. 4 illustrates the slope of the regression line drawn through the mean points was reduced in both 1 wk and 4 wk HT rats, compared with NT rats. The mean values of slope for regression fits of the individual curves in the NT group was 2.31 bpm mmHg SD 0.66 ; with a mean r2 of 0.971 SD 0.024 ; . In the 1-wk HT group, the mean slope was 1.30 bpm mmHg SD 0.38 ; P 0.031 vs. NT rats ; with a mean r2 of 0.975 SD 0.012 ; . In 4-wk HT rats the mean slope was 1.33 bpm mmHg SD 0.22 ; P 0.014 vs. NT rats ; with a r2 of 0.966 SD 0.020 ; . There was no difference in the slope of the regression line comparing 1 wk and 4 wk HT rats P .885 and anagrelide.
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Or microchannels in the endothelium of capillaries are large enough to allow drug molecules to penetrate unless the drug is highly protein bound in the blood.13 However, there are no antibiotics that have such high plasma drug protein binding to affect drug distribution enough to be clinically relevant. Tissues lacking pores or channels may inhibit penetration of some drugs discussed below and amevive.
Twenty-three of the 58 patients included in the efficacy analysis received rFIX for the prevention of intraoperative and postoperative bleeding in 30 surgical procedures Table 2 ; , including 12 permanent venous access placements, 7 circumcisions, 1 dental procedure, and 10 "other" procedures. Of these, 4 patients undergoing 5 surgical procedures received rFIX by continuous infusion regimens, and 19 patients received rFIX by bolus dosing only. Of all infusions administered for surgical procedures, 29 of 30 96.7% ; provided "excellent" or "good" hemostasis, and 1 of 30 3.3% ; was and anaprox.
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Forced by someone into doing something sexually while staying on the street, and having run away from home at age 12 or younger. They were also more likely to report having seen a health care provider since being on the streets, smoking tobacco, that most of the people they hang out with use needle exchange or bleach, having sex while drunk, having sex while high, and not using condoms 100% for vaginal sex with their primary partner. Multivariate Logistic Regression Analyses Predicting High-Risk Behaviors Logistic regression analyses were conducted on two outcomes defining high-risk behavior: ever injected drugs and inconsistent condom use. Age and gender were controlled for in both analyses. Variables found to be independently associated with inconsistent condom use defined as not using condoms 100% of the time for vaginal sex with primary partner ; are shown in Table 4. The best-fitting and androgel.
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