Bleomycin g2 phase

Functional mapping by Fos expression Many intense Fos-LI were found in various regions of the brain after icv administration of AM2 2 nmol rat ; Fig. 6, red dots on right side of each brain section ; . On the other hand, only a small number of Fos-LI was observed in the CNS after icv administration of saline Fig. 6, black dots on left side of each brain section ; . In the forebrain, many Fos-LI were localized to the piriform cortex Pir ; and the central amygdaloid nucleus CeA ; Fig. 7, F ; . In the hypothalamus, many Fos-LI were observed in the medial preoptic nucleus MPO ; , the SON Fig. 7, B ; , the PVN Fig. 7, D ; , the arcuate hypothalamic nucleus Arc ; , the periventricular regions of the third ventricle and the ventromedial hypothalamic nucleus VMH ; Fig. 7, H ; . In the midbrain and the brainstem, there were many Fos-LI in the locus coeruleus LC ; Fig. 7, J ; , the area postrema AP ; , the nucleus of the tractus solitarius NTS ; and the dorsal motor nucleus vagus DMV ; Fig. 7, L ; . On the other hand, only a few Fos-LI were observed in corresponding areas in controls injected icv with a vehicle Fig. 7, A, C, E, G, I and K. Carlson, R.W., Sikic, B.I., Turbow, M.M., and Ballon, S.S., " Cisplatin, Vinblastine, and Bleomycin PVB ; Therapy for Ovarian Germ Cell Tumors." Proc. Am. Soc. Clin. Onc 2: 15 1983. Nearly all residents have a GP, and GPs receive all medical correspondence from attending physicians regarding their patients. Questionnaires on specific cardiovascular diagnoses and risk factors were sent to the patients' GPs and or the last known attending physicians; 3, 035 questionnaires were sent out for 2, 293 patients. In total, 95% of the GPs responded, and 85% provided complete data. For 45% of the 5-year survivors, cardiovascular data were obtained from GPs only; for 39% of survivors, data were obtained from both medical records and the GP, and for 16% of survivors, data were obtained from the medical records alone. Uncertain cardiovascular diagnoses were verified through the patient's cardiologist n 52 ; . For patients who died from acute CVD without prior evidence of preceding CVDs, the date of death was recorded as date of CVD diagnosis, and the cause of death was recorded as the CVD diagnosis. One hundred seventy-three patients 7.4% ; were excluded from the original cohort because medical records could not be obtained and no information on CVD was received from the GP Fig 1 ; . The analytic cohort comprised 2, 339 5-year survivors. For 189 patients 8.1% ; , CVD information was not complete until at least January 1, 2000, the date of death, or the date of emigration. Treatment During the study period, treatment for TC after orchidectomy underwent significant changes. Seminoma Therapy for stage I and II seminoma included RT to the para-aortic and ipsilateral iliac lymph nodes in 86% of patients treated with RT. Prophylactic RT to the mediastinum and or left supraclavicular fossa was mainly applied in the period from 1972 to 1978 but has been gradually abandoned thereafter.19 Since the mid-1980s, irradiation has been further reduced to the para-aortic lymph nodes only, 19 and the RT dose has decreased from 30 to 26 Gy.17 Patients with disseminated seminoma were treated with cisplatin-based CT after 1979. Nonseminoma Initially, therapy for stage I and II nonseminoma included retroperitoneal lymph node dissection and or irradiation infradiaphragmatic with or without supradiaphragmatic ; with 40 to 50 Gy. Surveillance after orchidectomy became common beginning in 1985. Patients with disseminated nonseminoma have been treated with CT from the early 1970s, initially with single-agent dactinomycin20 or combinations of vinblastine and bleomycin.21 Beginning approximately in 1976, treatment consisted of four cycles of cisplatin, vinblastine, and bleomycin PVB ; followed by vinblastine maintenance therapy for 2 years.1 Maintenance therapy was omitted after 1984.22 In the mid-1980s, four cycles of bleomycin, etoposide, and cisplatin BEP ; became the standard treatment.23 Statistical Analysis A comparison was made between the incidence of CVDs in the study population and in the Netherlands male population using age-, sex-, and calendar periodspecific incidence rates for the period from 1972 through 2000 from the Continuous Morbidity Registration Nijmegen CMRN ; from several Netherlands GP practices. Comparison of recent incidence rates of coronary heart disease CHD ; , acute myocardial infarction MI ; , and angina pectoris AP ; from the CMRN with the incidence rates of several new registries in the Netherlands, which comprised only short-term incidence rates, showed that incidence rates of the CMRN were similar to the mean of all registries combined, indicating that the CMRN is representative of the Netherlands.24 Data on the incidence of MI, AP, heart failure HF ; , and cerebrovascular accidents CVAs ; were registered by CMRN, allowing for multiple separate diagnoses per person, but only the first of a given diagnosis was recorded.25, 26 Because our cohort only includes 5-year survivors, time at risk started 5 years from TC diagnosis and ended at the date of diagnosis of a specific cardiovascular event, emigration, death, or most recent information on CVD occurrence, whichever came first. The standardized incidence ratios SIRs ; of the observed and expected numbers of MI, AP, HF, CVA, and combined diagnostic groups in the study population were determined. The confidence intervals of the SIRs were calculated using exact Poisson probabilities of observed numbers. P values for tests for heterogeneity and for tests for trend were.

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