Daunorubicin fermentation
Treated with A + D. addition, there was a trend toward a either idarubicin 12 mg m2 A + I ; daunorubicin 45 greater CR rate and a significantly greater likelihood of mg m2 A + D ; daily for 3 days. A significantly higher CR achieving CR with one induction course of A I. Furtherrate was observed with A + I treatment, but no significant difference in response duration or survival was noted more, the adverse affect of hyperleukocytosis on response rate and survival was less evident with A + I treatment than between the two treatments. The three comparative American ~ t u ~suggest .'~~~ with treatment with A + D. that A I is better treatment for previously untreated An overview of idarubicin treatment for acute leukemia adults with AML than A + D. The CR rate was higher in all has recently been offered by Carella et al." Idarubicin and three studies for A + I and overall survival of the A I cytarabine have been studied in several schedules in pagroup was significantly better in two. tients with relapsed AML and the combination has been found to be effective with acceptable, expected to~icity.'~~'~-~'In a study of similar design to the present study, Arlin et aIz7 compared A + D the same dose and schedule as in Lambertenghi-Deliliers et alZZ treated 50 patients with the present study with mitoxantrone 12 mglm2 d daily for 3 previously untreated AML with idarubicin 12 mg mz d for days substituted for daunorubicin in the regimen A + M ; days, and cytarabine 240 mg m2for 7 days and observed Postremission therapy was of similar concept and design as an 80% CR rate. Subsequently, five trials, including the in the present study. The investigators concluded that, present study, were conducted to compare the relative because there were no significant differences between A + efficacy and toxicity of A + and A + D. two of the studies, only elderly patients were e n r ~there~ D and A + M with respect to CR rate or duration, survival, and ~ ~ or toxicity, A + D and A + M were comparable in the was no significant difference in outcome between the two first-line treatment of patients with AML. However, in that treatments, except that in the study by Mandelli et alZ3 a study a greater number of patients failed A D due to significantly greater number of CRs achieved CR with one persistant leukemia than did patients treated with A M, course of A + than with one course of A D, as the and patients treated with A + M were more likely to present study. The difficulty in showing an advantage for achieve CR with one induction course than were patients one treatment over another in elderly patients with AML treated with A + D. when such an advantage can be shown in younger patients It therefore appears that equitoxic A + I and A + M was noted by Bishop et a]." They could not show a survival regimens are better therapy than A + D for the treatment advantage for a three-drug regimen compared with a of newly diagnosed patients with AML. The present study two-drug regimen in older patients when a significant and others'83z6 suggest that A + I may be superior to A + survival advantage for the former was evident in younger because significant differences in outcome have been idenpatients. Our results are consistent with the Bishop et al" tified for A + I when directly prospectively compared with and Mandelli et alu data in that the superiority of A + and no significant differences were noted between compared with A + D was more evident in younger patients treated with A + D study of similar patients. design." A direct, prospective comparison of A D, A The results of the present study are essentially identical M, and A + I would be of interest and is warranted. to those of Berman et al, " who used cytarabine 200 mg mz as a 5-day continuous infusion together with three daily ACKNOWLEDGMENT bolus doses of idarubicin 12 mg m2 or daunorubicin 50 mg mz. In her study and ours, CR rate patients I50 years The following physicians also contributed patients to this study: of age ; , response duration, and survival were significantly Neil Abramson, Jacksonville, FL; James D. Ahlgren, Georgetown University, Washington, D C Phillip C. Amrein, Massachusetts better with A + I than with A + D. addition, in both General Hospital, Boston, M A Michael Auerbach, Franklin Square studies the majority of CRs to A + achieved a response Hospital, Baltimore, MD; James Bearden, University of South with one induction therapy course, whereas a minority of Carolina, Spartanburg, SC; Thomas M. Beck, Mountain States CRs to A D achieved remission with one course of Tumor Institute, Boise, ID; Marcus Black, Metairie, LA; William treatment. Furthermore, in both studies, hyperleukocytosis R. Friedenberg, Marshfield Clinic, Marshfield, WI; William Heim, unfavorably effected response rate in the A + D groups, but Scranton, PA; Khader Hussein, Oklahoma City, O K Rosaline R. not in the A + I groups. Mild hyperbilirubinemia was more Joseph, Medical College of Pennsylvania, Philadelphia, PA; Jacob common in the A + I group compared with the A + D Lokich, New England Deaconess Cancer Center, Boston, MA; group in both studies, and in both other toxicities of Mathew Luke, University Hospital, Jacksonville, FL; W. Angus Muir, Cancer Center of Virginia, Fredericksburg, VA; Martin induction therapy were similar in frequency and severity. Oken, Veterans Administration Medical Center, Minneapolis, Toxicity of postremission therapy is not discussed in BerMN; William Robinson, University of Colorado, Denver, CO; man et a1, " but in the present study postremission therapy Arnold Rubin, St Joseph's Hospital, Paterson, NJ; Richard Schuwith A + I was more myelosuppressive than A + D lof, George Washington University, Washington, DC; Richard T. treatment. The results of Berman et a1 and the present Silver, Cornell University Medical Center, New York, NY; Monica study suggest that idarubicin doses of 12 and 13 mg mZare B. Spaulding, Veterans Administration Medical Center, Buffalo, equivalent antileukemia doses, as are 45 and 50 mg mz NY; Phyllis A. Stephenson, Sarasota Oncology Center, Sarasota, daunorubicin doses. FL; Walter J. Stuckey, Tulane University Medical School, New In the study by Vogler et al" cytarabine was administered Orleans, LA; Mary L. Votaw, East Tennessee State University, at a dose of 100 mg m2by continuous intravenous infusion Johnson City, TN; and Galen L. Wampler, Virginia Commonwealth University, Richmond, VA. for 7 days as in the present study ; and combined with.
Discount Drugs
Table 3. Imputed Phenotypes and Risk of Colorectal Polyps
Current Treatment Results Approximately 50-75% of adults with AML achieve complete remission CR ; with the deoxycytidine analog cytarabine and an anthracycline antibiotic such as daunorubicin or idarubicin, or the anthracenedione mitoxantrone, which inhibit the enzyme topoisomerase IIa. 3.
Survival and Cell Kinetics Effects of Adriamycin on Mammalian Cells. Cancer Res., 33: 11-16, 1973. Bonadonna, G., Monfardini, S., DeLena, M., Fpssati-Bellani, F., and Beretta, G. Phase I and Preliminary Phase II Evaluation of Adriamycin NSC 123127 ; . Cancer Res., 30: 2572-2582, 1970. Bremerskov, V., and Linnemann, R. Some Effects of Daunomycin on the Nucleic Acid Synthesis in Synchronized L-Cells. European J. Cancer, 5: 317-330, 1969. Calendi, E., DiMarco, A., Regiani, M., Scarpinato, B., and Valentini, L. On Physicochemical Interactions between Dauno mycin and Nucleic Acids. Biochim. Biophys. Acta, 103: 25-49, 1965. Casazza, A. M., Di Marco, A., and Di Cuonzo, G. Interference of Daunomycin and Adriamycin on the Growth and Regression of Murine Sarcoma Virus Moloney ; Tumors in Mice. Cancer Res., 31: 1971-1976, 1971. Di Fronzo, G., Gambetta, R. A., and Lenaz, L. Distribution and Metabolism of Adriamycin in Mice. Comparison with Daunomycin. European J. Clin. Biol. Res., 16: 572-576, 1971. Di Marco, A., Gaetani, M., and Scarpinato, B. Adriamycin NSC-123, 127 ; . A New Antibiotic with Antitumor Activity. Cancer Chemotherapy Rept., 53: 33-37, 1969. Drewinko, B., Ho, D. H. W., and Barranco, S. C. The Effects of Arabinosylcytosine on Cultured Human Lymphoma Cells. Cancer Res., 32: 2737-2742, 1972. Drewinko, B., Lichtiger, B., and Trujillo, J. M. Synchronization of Cultured Human Lymphoid Cells. European J. Clin. Biol. Res., 18: 30-37, 1973. Drewinko, B., Novak, J. K., and Barranco, S. C. The Response of Human Lymphoma Cells in Vitro to Bleomycin and l, 3-Bis 2chloroethylH-nitrosourea. Cancer Res., 32: 1206-1208, 1972. Drewinko, B., Trujillo, J. M., and Gonzalez-Diddi, M. Harvesting Methods of Immunoglobulin Producing Cells. European J. Clin. Biol. Res., 16. 494-497, 1971. Isetta, A. M., Intini, C., and Soldati, M. On the Immunodepressive Action of Adriamycin. Experientia, 27: 202-204, 1971. Kim, S. H., and Kim, J. H. Lethal Effect of Adriamycin on the Division Cycle of HeLa Cells. Cancer Res., 32: 323-325, 1972. Madoc-Jones, H., and Mauro, F. Age Responses to X-rays, Vinca Alkaloids and Hydroxyurea of Murine Lymphoma Cells Synchro nized in Vivo. J. Nati. Cancer Inst., 45: 1131-1143, 1970. Massimo, L., Dagna-Bricarelli, F., and Fossati-Guglielmoni, A. Effects of Adriamycin on Human Lymphocytes Stimulated with PHA in Vitro. European J. Clin. Biol. Res., 75: 793-799, 1970. Middleman, E., Luce, J., and Frei, C., III. Clinical Trials with Adriamycin. Cancer, 28: 844-850, 1971. Oliverio, V. T. Pharmacology in the Chemotherapy Drug Develop ment Program of the National Cancer Institute. Cancer Chemo therapy Rept., 2 Part 3 ; : 73-79, 1971. 19. Razek, A., Valeriote, F., and Vietti, T. Survival of Hemopoietic and Leukemic Colony-forming Cells in Vivo following the Administration of Daunorubicin or Adriamycin. Cancer Res., 32: 1496-1500, 1972. Sandberg, J. S., Howsden, F. L., Di Marco, A., and Goldin, A. Comparison of Antileukemic Effect in Mice of Adriamycin with Daunomycin. Cancer Chemotherapy Rept., 54: 1-7, 1970. Silvestrini, R., Gambarucci, C., and Dasdia, T. Attivit Biologica delTAdriamicina in Vitro. Tumori, 56: 137-148, 1970. Trujillo, J. M., List-Young, B., Butler, J. J., Schullenberger, C. C., and Gott, C. Long Term Culture of Lymph Node Tissue from a Patient with Lymphocytic Lymphoma. Nature, 209: 310-312, 1966. Venditti, J. M. Treatment Schedule Dependency of Experimentally Active Antileukemic L1210 ; Drugs. Cancer Chemotherapy Rept. 2 Part 3 ; : 35-59, 1971. 24. Vig, B. K. Chromosome Aberrations Induced in Human Leuko cytes by the Antileukemic Antibiotic Adriamycin. Cancer Res., 31: 32-38, 1971. Wang, J. J., Chervinsky, D. S., and Rosen, J. M. Comparative Biochemical Studies of Adriamycin and Daunomycin in Leukemic Cells. Cancer Res., 52: 511-515, 1972. Wang, J. J., Cortes, E., Sinks, L., and Holland, J. F. Therapeutic Effect and Toxicity of Adriamycin in Patients with Neoplastic Disease. Cancer, 28: 837-843, 1971. Ward, D. C., Reich, E., and Goldberg, I. H. Base Specificity in the Interactions of Polynucleotides with Antibiotic Drugs. Science, 149: 1259-1263, 1965. Whang-Peng, J., Leventhal, B. G., Adamson, J. W., and Perry, S. The Effect of Daunomycin on Human Cells in Vitro and in Vivo. Cancer, 23: 113-121, 1969.
Daunorubicin medication
ALDESLEUKIN, PER SINGLE USE VIAL PROLEUKIN ; ARSENIC TRIOXIDE, 1 MG ASPARAGINASE, 10, 000 UNITS INJECTION, AZACITIDINE, 1 MG Vidaza ; INJECTION, CLOFARABINE, 1 MG Clolar ; BCG INTRAVESICAL ; PER INSTILLATION TICE BCG ; INJECTION, BEVACIZUMAB, 10 MG Avastin ; BLEOMYCIN SULFATE, 15 UNITS INJECTION, BORTEZOMIB, 0.1 MG Velcade ; CARBOPLATIN, 50 MG CARMUSTINE, 100 MG INJECTION, CETUXIMAB, 10 MG Erbitux ; CISPLATIN, POWDER OR S0LUTION, PER 10 MG CISPLATIN, 50 MG INJECTION, CLADRIBINE, PER 1 MG CYCLOPHOSPHAMIDE, 100 MG CYCLOPHOSPHAMIDE, 200 MG CYCLOPHOSPHAMIDE, 500 MG CYCLOPHOSPHAMIDE, 1.0 GRAM CYCLOPHOSPHAMIDE, 2.0 GRAM CYCLOPHOSPHAMIDE, LYOPHILIZED, 100 MG CYCLOPHOSPHAMIDE, LYOPHILIZED, 200 MG CYCLOPHOSPHAMIDE, LYOPHILIZED, 500 MG CYCLOPHOSPHAMIDE, LYOPHILIZED, 1.0 GRAM CYCLOPHOSPHAMIDE, LYOPHILIZED, 2.0 GRAM CYTARABINE LIPOSOME, 10 MG Depocyt ; CYTARABINE 100 MG CYTARABINE, 500 MG DACTINOMYCIN, 0.5 MG DACARBAZINE, 100 MG DACARBAZINE, 200 MG DAUNORUBICIN, 10 MG DAUNORUBICIN CITRATE, LIPOSOMAL FORMULATION, 10 MG DENILEUKIN DIFITOX, 300MCG DIETHYLSTILBESTROL DIPHOSPHATE, 250 MG DOCETAXEL, 20 MG TAXOTERE ; INJECTION, ELLIOTT'S B SOLUTION, 1 ML INJECTION, EPIRUBICIN HCL, 2 MG EPIRUBICIN HCL, 50MG ETOPOSIDE, 10 MG ETOPOSIDE, 100 MG. FLUDARABINE PHOSPHATE, 50 MG FLUOROURACIL, 500 MG FLOXURIDINE, 500 MG GEMCITABINE HCL, 200 MG GOSERELIN ACETATE IMPLANT, PER 3.6 MG ZOLADEX ; IRINOTECAN, 20 MG CAMPTOSAR ; IFOSFAMIDE, 1 GM MESNA, 200 MG IDARUBICIN HYDROCHLORIDE, 5 MG INJECTION, INTERFERON ALFACON-1, RECOMBINANT, 1 MCG INTERFERON, ALFA-2A, RECOMBINANT, 3 MILLION UNITS ROFERON A ; INTERFERON, ALFA-2B, RECOMBINANT, 1 MILLION UNITS INTERFERON, ALFA-N3, HUMAN LEUKOCYTE DERIVED ; , 250, 000 IU INTERFERON, GAMMA 1-B, 3 MILLION UNITS LEUPROLIDE ACETATE FOR DEPOT SUSPENSION ; , 7.5 MG LEUPROLIDE ACETATE, PER 1 MG LEUPROLIDE ACETATE IMPLANT, 65MG HISTRELIN, IMPLANT, 50MG Vantas ; MECHLORETHAMINE HYDROCHLORIDE, NITROGEN MUSTARD ; , 10 MG INJECTION, MELPHALAN HYDROCHLORIDE, 50 MG METHOTREXATE SODIUM, 5 MG METHOTREXATE SODIUM, 50 MG INJECTION, OXALIPLATIN, 0.5 MG INJECTION, PACLITAXEL PROTEIN-BOUND PARTICLES, 1 MG Abraxane ; PACLITAXEL, 30 MG PEGASPARGASE, PER SINGLE DOSE VIAL PENTOSTATIN, PER 10 MG PLICAMYCIN, 2.5 MG.
Free radic res commun, 1990, 11 1-3 ; , 127 - 36 one-electron reduction of daunorubicin intercalated in dna or in a protein: a gamma radiolysis study ; houee-levin c et al; the one-electron reduction of daunorubicin, an anthracycline antibiotic , intercalated in dna or in the apoprotein of the riboflavin binding protein, was studied by gamma radiolysis and deferasirox.
Relatively high daunorubicin concentrations could not restore that accumulation defect.
Note: This article was revised on January 12, 2007 to reflect that CR5459 was revised by CMS. The article was revised to reflect the new CR release date, transmittal number, and the Web address for accessing CR5459. All other information remains the same. * Provider Types Affected Physicians and other providers who bill Medicare contractors carriers, fiscal intermediaries FI ; , or Part A B Medicare administrative contractors A B MAC for professional services paid under the Medicare Physician Fee Schedule MPFS and delavirdine.
Conversion of daunorubicin to doxorubicin
3.4.1 Hepatic 3.4 Dose Modifications Bilirubin 20 - 50mol l reduce daunorubicin dose by 25% and reduce etoposide dose by 50%. Bilirubin 50mol l reduce daunorubicin dose by 50% and clinical decision whether to give etoposide Bilirubin 34mol l reduce cytarabine dose by 50.
Some covered drugs may have additional requirements or limits on coverage. These requirements and limits may include: Prior Authorization: First UA Medicare Group Part D requires you or your physician to get prior authorization for certain drugs. This means that you will need to get approval from First UA Medicare Group Part D before you fill your prescriptions. If you don't get approval, First UA Medicare Group Part D may not cover the drug. Quantity Limits: For certain drugs, First UA Medicare Group Part D limits the amount of the drug that First UA Medicare Group Part D will cover. For example, First UA Medicare Group Part D provides 34 tablets per prescription for LIPITOR. This may be in addition to a standard one month or three month supply. Step Therapy: In some cases, First UA Medicare Group Part D requires you to first try certain drugs to treat your medical condition before we will cover another drug for that condition. For example, if Drug A and Drug B both treat your medical condition, First UA Medicare Group Part D may not cover drug B unless you try Drug A first. If Drug A does not work for you, First UA Medicare Group Part D will then cover Drug B. You can find out if your drug has any additional requirements or limits by looking in the formulary that begins on page 7. You can ask First UA Medicare Group Part D to make an exception to these restrictions or limits. See the section, "How do I request an exception to the First UA Medicare Group Part D's formulary?" below for information about how to request an exception and demeclocycline.
For vitamin because of of this vitamin.
Because the parent drug daunorubicin is a vesicant this liposomal form is treated with equal caution and desipramine.
Measure represented a ratio of this baseline value. Neutrophil yield per animal was between 3 and 10 106. A significant main effect of the drug was detected using analysis of variance P .001 ; . The Bonferroni multiple comparison test revealed that neutrophil receptor detection in the 2 and 10 hours, without drug groups was significantly increased compared with the 2 and 10 hours, with drug groups P .05 ; Figure 1 ; . SKIN INFILTRATION NEUTROPHIL COUNTS Neutrophil infiltration was significantly reduced in the treated vs untreated 2-hour ischemic groups 2 hours, with drug vs 2 hours, without drug; P .05 ; and the 10hour ischemic groups 10 hours, with drug vs 10 hours, without drug; P .01 ; Figure 2!
Daunorubicin will decrease your body's ability to fight infections and dexedrine.
Daunorubicin tablet
The following guidelines apply when billing for chemotherapy filed on a CMS-1450 claim form: Acute Care Injections Use Revenue Code 0331 with HCPCS Code Q0083 for the administration fee. Use Revenue Code 0636 with one or more of the most appropriate CPT or HCPCS code for the drug when filing the injection of chemotherapy. Note: Code Q0083 reimburses the facility for the administration fee, whereas the CPT or HCPCS code reimburses the facility for the drug. Oral Dosage Use Revenue Code 0332 with HCPCS Code Q0083 for the administration fee. Use Revenue Code 0636 with one or more of the most appropriate CPT or HCPCS code for the drug when filing the oral dosage of chemotherapy. See St. Anthony's CMS-1450 Editor for determination of the most appropriate CPT or HCPCS code. ; Note: Code Q0083 reimburses the facility for the administration fee, whereas the CPT or HCPCS code reimburses the facility for the drug. IV Use Revenue Code 0335 with HCPCS Code Q0084 or Q0085 for the administration fee. Use Revenue Code 0636 with one or more of the appropriate J Codes for the drug when filing for the IV of chemotherapy. See St. Anthony's CMS 1450 Editor for determination of most appropriate CPT or HCPCS code
Pot Distillers Yeast 18% can ferment fruit brandy, fruit schnapps, grappa, brandy, and other mashes to 18% while it greatly improves taste after distillation, and gives a better yield. It is a hit with all schnapps distillers. It can sometimes be used to ferment larger batches to 18% alcohol by using one sachet to 50 liters of sugar or fruit mash half dosage ; . This is normally impossible with high alcohol Turbos because they ferment violently and produce so much heat early in the process that the yeast dies and dextroamphetamine.
1. Wear white elastic TED hose FIRST 2 WEEKS--BOTH LEGS AT ALL TIMES WEEKS 3-5--WEAR ONLY DURING THE DAY, SURGICAL LEG ONLY. AFTER 5 WEEKS DO NOT NEED TO WEAR THEM ANYMORE and daunorubicin.
19. Glossmann JP, Josting A, Diehl V. New treatments for Hodgkin's disease. Curr Treat Options Oncol 2002; 3: 283 Barth S, Huhn M, Matthey B, Tawadros S, Schnell R, Schinkothe T, et al. Ki-4 scFv ; -ETA', a new recombinant anti-CD30 immunotoxin with highly specific cytotoxic activity against disseminated Hodgkin tumors in SCID mice. Blood 2000; 95: 3909 Alexandrakis MG, Passam FH, Kyriakou DS, Bouros D. Pleural effusions in hematologic malignancies. Chest 2004; 125: 1546 Haluska FG, Brufsky AM, Canellos GP. The cellular biology of the Reed-Sternberg cell. Blood 1994; 84: 1005 Drexler HG, Leber BF, Norton J, Yaxley J, Tatsumi E, Hoffbrand AV, et al. Genotypes and immunophenotypes of Hodgkin's disease-derived cell lines. Leukemia 1988; 2: 371 Kamesaki H, Fukuhara S, Tatsumi E, Uchino H, Yamabe H, Miwa H, et al. Cytochemical, immunologic, chromosomal, and molecular genetic analysis of a novel cell line derived from Hodgkin's disease. Blood 1986; 68: 285 Schaadt M, Diehl V, Stein H, Fonatsch C, Kirchner HH. Two neoplastic cell lines with unique features derived from Hodgkin's disease. Int J Cancer 1980; 26: 723 Diehl V, Kirchner HH, Burrichter H, Stein H, Fonatsch C, Gerdes J, et al. Characteristics of Hodgkin's disease-derived cell lines. Cancer Treat Rep 1982; 66: 615 Kuppers R, Klein U, Schwering I, Distler V, Brauninger A, Cattoretti G, et al. Identification of Hodgkin and ReedSternberg cell-specific genes by gene expression profiling. J Clin Invest 2003; 111: 529 Dukers DF, van Galen JC, Giroth C, Jansen P, Sewalt RG, Otte AP, et al. Unique polycomb gene expression pattern in Hodgkin's lymphoma and Hodgkin's lymphoma-derived cell lines. J Pathol 2004; 164: 873 Kuppers R, Schmitz R, Distler V, Renne C, Brauninger A, Hansmann ML. Pathogenesis of Hodgkin's lymphoma. Eur J Haematol 2005 Suppl ; : 26 33. 30. Kis LL, Nishikawa J, Takahara M, Nagy N, Matskova L, Takada K, et al. In vitro EBV-infected subline of KMH2, derived from Hodgkin lymphoma, expresses only EBNA-1, while CD40 ligand and IL-4 induce LMP-1 but not EBNA-2. Int J Cancer 2005; 113: 937 Kis LL, Nagy N, Klein G, Klein E. Expression of SH2D1A in five classical Hodgkin's disease-derived cell lines. Int J Cancer 2003; 104: 658 Markasz L, Stuber G, Flaberg E, Gustafsson Jernberg A, Eksborg S, Olah E, et al. Cytotoxic drug sensitivity of EpsteinBarr virus transformed lymphoblastoid B cells. BMC Cancer 2006; 6: 265. Fogli S, Danesi R, Gennari A, Donati S, Conte PF, Del Tacca M. Gemcitabine, epirubicin and paclitaxel: pharmacokinetic and pharmacodynamic interactions in advanced breast cancer. Ann Oncol 2002; 13: 919 Andersson B, Andersson I, Beran M, Ehrsson H, Eksborg S. Liquid chromatographic monitoring of daunorubicin and daunorubicinol in plasma from leukemic patients treated with daunorubicin or the daunorubicin-DNA complex. Cancer Chemother Pharmacol 1979; 2: 15 Toffoli G, Corona G, Cattarossi G, Boiocchi M, Di Gennaro G, Tirelli U, et al. Effect of highly active antiretroviral therapy HAART ; on pharmacokinetics and pharmacodynamics of doxorubicin in patients with HIV-associated nonHodgkin's lymphoma. Ann Oncol 2004; 15: 1805 Kato Y, Nishimura S, Sakura N, Ueda K. Pharmacokinetics of etoposide with intravenous drug administration in children and adolescents. Pediatr Int 2003; 45: 74 and dextromethorphan.
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