Too much fluoride during pregnancy
Venkatestan P, Gladman J, McFarlane JT, et al. A hospital study of community-acquired pneumonia in the elderly. Thorax 1990; 45: 254258. Riquelme R, Torres A, El-Ebiary M, et al. Communityacquired pneumonia in the elderly: etiology, risk, and prognostic factors. J Respir Crit Care Med 1996; 154: 14501455. British Thoracic Society, the Public Health Laboratory Service. Community-acquired pneumonia in adults in British hospitals in 19821983: a survey of aetiology, mortality, prognostic factors and outcome. Q J Med 1987; 239: 195220. Ewig S, Bauer T, Hasper E, et al. Prognostic analysis and predictive rule for outcome of hospital-treated community-acquired pneumonia. Eur Respir J 1995; 8: 392 Karalus NC, Cursons RT, Leng RA, et al. Community acquired pneumonia: aetiology and prognostic index evaluation. Thorax 1991; 46: 413418. Farr BM, Sloman AJ, Fisch MJ. Predicting death in patients hospitalized for community acquired pneumonia. Ann Intern Med 1991; 115: 428436. Neill AM, Martin IR, Weir R, et al. Community-acquired pneumonia: aetiology and usefulness of severity criteria on admission. Thorax 1996; 51: 10101016. Woodhead M. Predicting death from pneumonia. Thorax 1996; 51: 970971. Fine MJ, Auble TE, Yealy DM, et al. A prediction rule to identify low-risk patients with community-acquired pneumonia. N Engl J Med 1997; 336: 243250. Carson CA, Fine MJ, Smith MA, et al. Quality of published reports of the prognosis of community-acquired pneumonia. J Gen Intern Med 1996; 9: 1319. Woodhead M, Torres A. Definition and classification of pneumonia. Eur Respir Mon 1997; 3: 312. Fine MJ, Smith MA, Carson CA, et al. Prognosis and outcome of patients with community-acquired pneumonia: a meta-analysis. JAMA 1996; 275: 134141. Fein AM, Niederman MS. Severe pneumonia in the elderly. Clin Geriatr Med 1994; 10: 121141. Ewig S. Community-acquired pneumonia: epidemiology, risk, and prognosis. Eur Respir Mon 1997; 3: 1335.
Controls Figure 4A and B ; . The dermal melanocytes appeared normal in their distribution. Although there appeared to be an increase in the number of melanocytes lining the dermoepidermal junction in focal areas on FontanaMassonstained sections, this increase was not evident when melanocytes were counted on S100-stained sections. A few CD3-positive T lymphocytes and CD68-positive macrophages were noted in both groups, with great variability in the counts Table ; . Intracytoplasmic melanin granules were absent in the CD68-positive cells and were only seen in the S100-positive cells, suggesting that the melanin granules were present within the melanocytes and were not engulfed by tissue macrophages!
Use of fluoride and avoidance of cariogenic foods. Unexplained dental caries, especially in root and incisal sites, may be the first apparent clinical sign in Sjgren's syndrome.49.
Evan Spivack, DDS, FAGD "The only thing that remains constant is change." How many times have we heard this phrase and how true it is, especially in today's fast moving and evolving world of dentistry. This saying has become more than just an entry in a book of quotations; it reflects the way we view ourselves and the way we see the world we live in. Humans like the comfort of the familiar and the constant, but we must deal with situations that are ever-shifting in all aspects of our lives. In our personal lives, we see change on a daily basis: we age, we go from childhood to adulthood and hopefully we gain strength and wisdom. Our views of the world change with our educational and personal experiences, and these changes, these new thoughts and opinions, in turn help to reshape the world. As dentists, we experience many changes as our careers progress. We are first students, formally in school, then in residencies and finally we are in our practices. As we grow, we become teachers which is the very definition of the word doctor ; , perhaps formally as members of a faculty or informally to younger dentists we encounter in practice. As we become proficient in our profession, we again become students, turning to continuing education to learn new skills that will enhance and change our pattern of practice. It is important that we, as dentists, embrace change. We must remain current in our knowledge of new materials and techniques, we must stay abreast of the latest technology. In our role as health care professionals, we must also keep up with relevant medical information that will impact on the care of our patients. We should not necessarily be "first on the block" to jump at something new, but neither should we be the last to embrace concepts and products that have passed scientific muster. Many of us become too comfortable with the way in which we practice, the materials we use and the ideas we espouse. We turn away from new ideas almost by reflex, and find ways to rationalize our outmoded beliefs. The challenge against complacency, if we truly wish to do the best that we can for our patients and ourselves, must be a never-ending one. In that vein, I wish to address a significant change for NJAGD and Wisdom. To better serve our membership, the NJAGD Board has selected a new publisher for our journal. Mr. Ettore Palmeri has a winning track record of success in the dental publishing world with dental journals such as ours. Personally I looking forward to work with Ettore to improve the content and the reproduction quality of Wisdom. As we look forward with anticipation, I also wish to look back, and offer thanks to NJDA's Eric Elmore, who helped elevate Wisdom so dramatically over the past few issues. I have no doubt but that Eric will continue to play an important role in the continuing growth of NJAGD as we continue to evolve into the future.
Stannous fluoride in food
Clin Exp Dermatol 1983; 8: 46376. Jedele KB, Michels VV. Familial dermographism. J Med Genet 1991; 39: 2013. Kaplan AP. Unusual cold-induced disorder: cold dependent dermographism and systemic cold urticaria. J Allergy Clin Immunol 1984; 73: 4536. Kontou-Fili K, Borici-Mazi R, Kapp A, et al. Physical urticaria: classification and diagnostic guidelines. An EACCI position paper. Allergy 1997; 52: 50413. James J, Warin RP. Factitious wealing at the site of previous cutaneous response. Br J Dermatol 1969; 81: 8824. Smith JA, Mansfield LE, Fokakis AG, Nelson HS. Dermographia caused by IgE mediated penicillin allergy. Ann Allergy 1983; 51: 302. Warin RP. Clinical observations on delayed pressure urticaria. Br J Dermatol 1989; 121: 2258. Barlow RJ, Warburton F, Watson K, et al. Diagnosis and incidence of delayed pressure urticaria in patients with chronic urticaria. J Acad Dermatol 1993; 29: 9548. Dover JS, Black AK, Ward AM, Greaves MW. Delayed pressure urticaria. Clinical features, laboratory investigations, and response to therapy of 44 patients. J Acad Dermatol 1988; 18: 128998. Greaves MW. The physical urticarias. Clin Exp Allergy 1991; 21: 2849. Illig L, Kunick J. [Clinical picture and diagnosis of physical urticaria. I.] Hautarzt 1969; 20: 16778. Lawlor F, Black AK, Breathnach AS, Greaves MW. Vibratory angioedema: lesion induction, clinical features, laboratory and ultrastructural findings and response to therapy. Br J Dermatol 1989; 120: 939. Neitaanmaki H. Cold urticaria. Clinical findings in 220 patients. J Acad Dermatol 1985; 13: 63644. Ormerod AD, Kobza-Black A, Milford-Ward A, Greaves MW. Combined cold urticaria and cholinergic urticaria-- clinical characterization and laboratory findings. Br J Dermatol 1988; 118: 6217. Constanzi JJ, Coltman CA Jr. Kappa chain cold precipitable immunoglobulin G IgG ; associated with cold urticaria. I. Clinical observations. Clin Exp Immunol 1967; 2: 16778. Daman L, Lieberman P, Ganier M, Hashimoto K. Localized heat urticaria. J Allergy Clin Immunol 1978; 61: 2738. Juhlin L, Malmros-Enander I. Solar urticaria: mechanism and treatment. Photodermatol 1986; 3: 1648. Harber LC, Whitman GB, Armstrong RB, Deleo VA. Photosensitivity diseases related to interior lighting. Ann N Y Acad Sci 1985; 453: 31727. Ravits M, Amstrong RB, Harber LC. Solar urticaria. Clinical features and wavelength dependence. Arch Dermatol 1982; 118: 22831. Hirschmann JV, Lawlor F, Louback JB, et al. Cholinergic urticaria. A clinical and histological study. Arch Dermatol 1987; 123: 4627. Berth-Jones J, Graham-Brown RA. Cholinergic pruritus, erythema and urticaria: a disease spectrum responding.
Cumulative effects the primary degradation product of sulfuryl fluoride is fluoride and fluphenazine.
Dead layer of skin; however, efforts should be taken to prevent ingestion, inhalation, or absorption through broken skin. 3 ; Cesium-137. Cesium-137 137Cs ; is used in the soil density and moisture tester Campbell Pacific Model MC-1 ; . It has a 10 mCi 137Cs source located in a probe tip used to determine the density of the soil at a construction site. The 137Cs emits a negative beta particle as it decays to 137Ba, which in turn decays by emitting gamma rays. The hazard is minimal since the radioactive source is totally shielded in double encapsulated stainless steel. 4 ; Promethium-147. Promethium-147 147Pm ; has a half-life of 2.64 years and is a beta particle emitter used in luminous paints. The only weapon system that uses this element is the M72 series, 66 mm, light antitank weapon LAW ; . For over 30 years, the LAW was manufactured with 147Pm. Promethium-147 is located on the 100 m and 150 m range markers on the sight. In the early 1980's, the "E" version of the LAW's sight was changed, eliminating the need for 147Pm. However, the older models of the LAW, M72, M72A1, and M72A2, still remain in use for training and are therefore still in long-term storage. There are no known hazards involved with 147Pm. 5 ; Radium-226. Radium-226 226Ra ; was used on the faces and pointers of dials and gauges of instruments of tactical combat vehicles. Radium dials and radium marks on toggle switches may also be found in some radios of the older VRC-12 series, including the AN GRC-19, the VRC-46, and the GRC-106. Radium-226 is primarily an alpha emitter, but it also emits some low energy beta and gamma radiation. The hazard was such that these commodities are no longer in use nor in DOD storage depots, and they have not been procured since 1969. Nonradioactive replacements have been available in the supply system for all of these radioactive items. 6 ; Thorium-232. Thorium-232 232Th ; is a naturally occurring radioisotope of thorium and is an alpha emitter. When thorium is heated in air, it glows with a white light. For this reason, one of the major uses of thorium has been the Welsback lantern mantle used in portable gas lanterns. Thorium-232 is also in radiac sets AN VDR-2, AN PDR-54, and the AN PDR-77. Thermal optic fire control systems have a multilayer, infrared, antireflective coating that contains 232Th as a fluoride compound thorium fluoride ; . Thorium-coated optics are found on many night vision devices, such as the AN TAS-4 series. Also, natural thorium oxide evenly dispersed in simple nickel thorium alloy is used in the combustor liner for the Abrams series tank turbine engine because the alloy can withstand a great amount of heat. In general, 232Th presents a minimal hazard, but care should be taken to avoid inhalation or ingestion of any particles from damaged components that contain 232Th. 7 ; Americium-241. Americium-241 241Am ; does not occur naturally; it is a daughter product of the decay of 241P and has a half-life of 458 years. Americium-241 is used as a sealed source in the M43A1 Chemical Agent Detector that is a component of the M8A1 alarm. Americium-241 is primarily an alpha and a very low energy gamma emitter. External exposure is not a concern unless large amounts of the 241Am are located in one area and personnel are in close contact for an extended period of time. The high energy alpha emission can present an internal radiation hazard if ingested. Americium-241 is chemically analogous to calcium and it can replace.
Process of removing fluoride from water
And the upper stratum spinosum reacted distinctly stronger than in the other mammals Table A3, Fig. 6b ; . With regard to the mammalian skin glands, the apocrine tubular glands, particularly, showed clearly positive reaction staining in the secretory cells and the luminal secretion Table A4, Fig. 6a ; . Based on the lectin reactions, the integumental free sugar spectrum in the different vertebrate species studied was composed as follows: 1 ; fishes: epidermis with -D-mannose specific lectin: Con A ; , -D-Nacetylglucosamine WGA, GSA-II ; , -D-N-acetylgalactosamine DBA, SBA ; , -D-galactose GSA-I ; , -Dgalactose BPA, PNA ; , -L-fucose UEA-I ; , and sialic acids LFA 2 ; amphibians: epidermis with -Dmannose Con A ; , -D-N-acetylglucosamine WGA, GSA-II ; , -D-galactose GSA-I ; , -D-galactose BPA, PNA ; , and -L-fucose UEA-I ; , and dermal glands with -D-N-acetylglucosamine WGA, GSA-II ; , -D-N and flurazepam.
Figure 8. Cryptosporidiosis. 45-year-old man with microbiologically proven cryptosporidiosis. There are multiple loops of fluid-filled small bowel showing concentric wall thickening. Imaging, Volume 14 2002 ; Number 1.
Phenylmethanesulfonyl fluoride PMSF, 30 tM ; , 0.1%, v v ; . The suspension was maintained at mm [31] with occasional shaking and centrifuged centrifuge ; . The supernatants were mixed with a in Tris-HC1 NaC1 buffer, of WGA-Sepharose Mixing was achieved by using a rotary agitator temperature ; . The beads with the Tris-HC1 NaC1 were eluted by treatment lyzed by SDS-PAGE. The slices that were solubilized night ; . Scintillation fluid counted and flurbiprofen.
What is fluoride in
The R.W. Johnson Pharmaceutical Research Institute, Toronto, Canada Accepted for publication January 8, 1999 This paper is available online at : jpet.
50. Hsu, S.Y., Kaipia, A., McGee, E., Lomeli, M. and Hsueh, A.J. 1997 ; Bok is a pro-apoptotic Bcl-2 protein with restricted expression in reproductive tissues and heterodimerizes with selective anti-apoptotic Bcl-2 family members. Proc. Natl Acad. Sci. USA, 94, 1240112406. 51. Zhang, H., Holzgreve, W. and De Geyter, C. 2000 ; Evolutionarily conserved Bok proteins in the Bcl-2 family. FEBS Lett., 480, 311313. 52. Matsuyama, S., Schendel, S.L., Xie, Z. and Reed, J.C. 1998 ; Cytoprotection by Bcl-2 requires the pore-forming 5 and 6 helices. J. Biol. Chem., 273, 3099531001. 53. Ottilie, S., Diaz, J.L., Chang, J., Wilson, G., Tuffo, M.K. Weeks, S., McConnell, M., Wang, Y., Oltersdorf, T. and Fritz, L.C. 1997 ; Structural and functional complementation of an inactive Bcl-2 mutant by Bax truncation. J. Biol. Chem., 272, 1695516961. 54. Yin, X.M., Oltvai, Z.N. and Korsmeyer, S.J. 1994 ; BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax. Nature, 369, 321323. 55. Shibasaki, F., Kondo, E., Akagi, T. and McKeon, F. 1997 ; Suppression of signalling through transcription factor NF-AT by interactions between calcineurin and Bcl-2. Nature, 386, 728731. 56. Wang, H.G. and Reed, J.C. 1998 ; Bc1-2, Raf-1 and mitochondrial regulation of apoptosis. Biofactors, 8, 1316. 57. Altschul, S.F., Madden, T.L., Schaffer, A.A., Zhang, J., Zhang, Z., Miller, W. and Lipman, D.J. 1997 ; Gapped BLAST and PSI-BLAST: a new generation of protein database search programs. Nucleic Acids Res., 25, 33893402. 58. Lennon, G., Auffray, C., Polymeropoulos, M. and Soares, M.B. 1996 ; The I.M.A.G.E. Consortium: an integrated molecular analysis of genomes and their expression. Genomics, 33, 151152. 59. Corpet, F. 1988 ; Multiple sequence alignment with hierarchical clustering. Nucleic Acids Res., 16, 1088110890. 60. Hofmann, K. and Stoffel, W. 1993 ; TMbase a database of membrane spanning proteins segments. Biol. Chem., 347, 166 and fluvastatin.
Sodium fluoride density
LOAEL-to-NOAEL extrapolation ; , to account for uncertainties with predicting chronic exposure effects on the basis of subchronic exposure studies subchronic to chronic extrapolation ; , and to address uncertainties when the database on the chemical is inadequate. Sometimes a modifying factor is used to account for additional uncertainty not addressed by the standard uncertainty factors. Typically, uncertainty factors are assigned values ranging from 1 to 10. If information about a factor is sparse and uncertainty is high, a default value of 10 is generally used. If information is available, the uncertainty factor might be reduced to 1. For an uncertainty factor that falls between 1 and 10, a factor of 3 is typically assigned, because 3 is the approximate logarithmic mean of 1 and 10, and it is assumed that the uncertainty factor is distributed lognormally EPA 1994 ; . To calculate a reference dose, the NOAEL or LOAEL is divided by the product of the uncertainty factors. EPA typically uses a maximum of 3, 000 for the product of four uncertainty factors that individually are greater than 1 and a maximum of 10, 000 with five uncertainty factors Dourson 1994 ; . More recently, the benchmark dose is being used as the starting point for calculating reference doses. The benchmark dose is a dose with a specified low level of excess health risk, generally in the range of 1% to 10%, which can be estimated from data with little or no extrapolation outside the experimental dose range. Specifically, the benchmark dose is derived by modeling the data in the observed experimental range, selecting an incidence level within or near the observed range e.g., the effective dose producing a 10% increased incidence of response ; , and determining the upper confidence limit on the model. To account for experimental variation, a lower confidence limit or uncertainty factors on the benchmark dose are used to ensure that the specified excess risk is not likely to be exceeded. To derive an MCLG, the reference dose is multiplied by a typical adult body weight of 70 kg and divided by an assumed daily water consumption of 2 L yield a drinking water equivalent level. That level is multiplied by a percentage of the total daily exposure contributed by drinking water usually 20% ; to calculate the MCLG. EPA then uses the MCLG to set an enforceable standard the MCL ; . The MCL is set as close to the MCLG as feasible. Carcinogenic Contaminants EPA sets MCLGs of zero for contaminants that are known or probable human carcinogens. For chemicals judged to be possibly carcinogenic to humans, EPA has recently begun applying an uncertainty factor between 1 and 10 to the reference dose derived from noncancer health effects to determine some exposure standards, such as certain ambient waterquality criteria EPA 2000d ; . EPA stipulates that the water concentrations estimated to result in 1 10-6 to 1 10-5 excess cancer risks should also be compared with the reference dose. NEW RISK ASSESSMENT CONSIDERATIONS Since the fluoride MCLG and SMCL were originally issued, there have been a number of developments in risk assessment. A few of those issues were described above in the discussion of current risk assessment practices e.g., use of benchmark dose ; . Below, a few specific issues relevant to the committee's review of the drinking water standards for fluoride are discussed.
Pro strong fluoride nail treatment
| Fluoride detox boronToothbrushing with fluoride toothpaste 1500 ppm ; at least 1 min, 2-3 times day, plus fluoride rinsing 0.05% NaF ; 4 times daily or 4 fluoride tablets 0.25 mg F ; daily or 4 fluoride chewing gum pieces 0.25 mg F ; daily continue as long as caries activity is high and focalin.
Site a monitored two different sets of metrics: number of low back pain patients and visits and number of visits per patient, total and per clinic, using ads data presence of documentation form 695-r, documentation of referral to back class, and documentation that the red flags had been checked, using review of a sample of low back pain patients' charts.
Received August 20, 1998; final revision received December 11, 1998; accepted December 11, 1998. From the Stroke Clinic, Stroke Program, Instituto Nacional de Neurologia y Neurocirugia, Mexico City, Mexico. Reprint requests to Fernando Barinagarrementeria, MD, Stroke Clinic, Instituto Nacional de Neurologia y Neurocirugia, Insurgentes Sur 3877, Tlalpan, 14269, Mexico City, Mexico. E-mail fbarinaga compuserve 1999 American Heart Association, Inc. Stroke is available at : strokeaha and follistim.
| Any antiveratrine action. Since the increase in potassium concentration of coronary sinus blood was found to be largely nonspecific in nature the correlation of antiveratrine and antiarrhythinic actions of drugs on the basis of atrial arrhythmias cannot be explained by the action of these drugs on efflux of potassium from the skeletal and cardiac muscle. There is no correlation between the antiarrhythmic and antiveratrine action of drugs on the basis of ventricular arrhythnias alone. The probable mechanisms responsible for this discrepancy are discussed and fluoride.
Prerequisite: successful completion of anat 125 and satisfactory completion of chem 143 or one year of high school chemistry with grade of a or 4051 uyeshiro s 12: 40 - 01: 35 tr msci 208 and and 4060 vos l and and 3999 ward d and and 4000 ward d and and 12: 15 - 02: 15 11: - 12: 10 12: - 01: 35 09: 00 - 11: 00 11: 00 - 11: 55 05: 00 - 05: 55 02: - 04: 40 01: - 02: 40 05: 00 - 05: 55 06: - 07: 10 07: - 09: 10 mw mw msci 223 msci 223 msci 208 msci 223 msci 223 msci 208 msci 233 msci 223 msci 208 msci 223 msci 223 and formoterol.
Mother's earthly home and gave up his body for us, on the Cross and in the Eucharist through which we become one with his flesh." Professor West went on to say that the Pope spoke of the Theology of the Body the study of God in the body ; because the body alone is capable of making visible what is invisible -- namely, the image and likeness of God in each of us. The deepest mystery of God is the eternal exchange of love that is the Trinity, and this too is imaged for us in the body, especially in the self-giving, sexual union of a man and a woman. Marriage is a sacrament because it is a sign, a foretaste of the eternal marriage. The sign and symbol of this is the sexual union through which two become one and through which new life is created. "This explains why we're all so interested in sex, because we are looking for God, " West said. He recalled G.K.Chesterton saying that every man who knocked on a brothel door was looking for God; he was just confused about where to look. The human sexual union was creat.continued on p2.
Fluoride kenya
Acids is downstream from the guanine nucleotide and, hence, at the catalytic unit. This would also tend to indicate that these fatty acid changes are functionally affecting, in this case, the inner hemileaflet of the plasma membrane. This is in agreement with the work of Alam, Ren and Alam 12 ; in the heart. In contrast with our data and others' 12 ; of higher hormone-stimulated adenylate cyclase activity in fish oil- and corn oil-fed groups, it has been reported that dietarily induced high levels of linoleic acid 10, 11 ; and linolenic acid 11 ; in liver plasma membrane de creases glucagon-stimulated adenylate cyclase activity. These discrepancies may be due to the differences in the quantity and or the type of fat used. Their studies utilized 20% by weight ; fat diets and would be con sidered high fat diets compared to the 10% fat used in the present study. High fat diets might have altered the physiological state of the animals and thus modified the levels of circulating hormones. In addition, the fish oil used in our study is rich in C20: 5 n-3 ; EPA ; and C22: 6 n-3 ; DHA ; , but not C18: 3 n-3 ; linolenate ; . We observed an increased incorporation of EPA and DHA in liver plasma membrane 22 ; . These two fatty acids may have specific effects on the catalytic unit of the adenylate cyclase complex. Colard, Kervabon and Roy 37 ; have studied the effect in vitro of linoleate on aden ylate cyclase of rat liver plasma membrane. They found an increase in basal, fluoride-, and glucagon-stimulated adenylate cyclase activity in rat liver plasma membrane with increasing membrane linoleate. Prostaglandin E! PGEJ-stimulated adenylate cyclase activity was also found to increase in mouse LM cells grown in a high 18: 2 n-6 ; medium 38 ; . These findings are consistent with our observations. Given the fact that we observed no changes in cholesterol phospholipid molar ratio or membrane fluidity by fluorescence polarization 22 ; with these three treatments, it can be hypothesized that these effects are due to fatty acid changes. However, the exact rationale for these effects remains unclear. Since the major change in the membranes of C-fed versus B-fed rats is the total n-6 ; [ 16: l n-7 ; + 18: l n-9 ; ] ratio, while the liver plasma membranes from F-fed rats showed changes in longer-chain, more unsaturated fatty acids, it remains to be determined whether the effect is due to chain length, degree of unsaturation, or exact fatty acid positioning in particular phospholipids. The effect of dietary cholesterol is more difficult to interpret than that of dietary fatty acids. Dietary cho lesterol depressed activity in livers from C CH- and F CH-fed rats relative to their non-supplemented coun terparts for glucagon fold stimulation Fig. 1 ; , glucagon specific activity Fig. 2b], and fluoride specific activity Fig. 3u ; . The activity in livers from B-fed animals was already at what may be considered a minimal level and, if anything, increased slightly with cholesterol feeding. It would appear that there is an impairment of either Gs-protein or the coupling process with cholesterol feeding. However, besides increases in membrane cho and forteo.
Fluoride overdose child
Congratulations to Richard Stanford who has successfully defended his Master's thesis entitled The English Channel: a mixed fishery, but which mix is best? Richard came to the Fisheries Centre after completing his Bachelor of Science at the University of Southampton, England, in 2000. His thesis involved building ecosystem models of the English Channel so that he could study the effects of climate and different fishing policies on the species composition of the region. Richard has returned to England and will be preparing his thesis for publication. Watch the Fisheries Centre Research Reports on the Fisheries Centre website fisheries.ubc ; for more on Richard's work and fluphenazine.
Fluoride trays for cancer patients
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Potentiometric determination of fluoride in toothpaste
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