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Four patients used 800mg indinavir twice daily plus 100mg ritonavir twice daily; five patients used 1200mg indinavir twice daily; one patient used 1200mg indinavir twice daily in combination with 100mg ritonavir twice daily.
1. Culbertson WW, Huang AJ, Mandelbaum SH, Pfugfelder SC, Boozalis GT, Miller D. Effective treatment of phlyctenular keratoconjunctivitis with oral tetracycline. Ophthalmology. 1993; 100: 1358-1366. Meisler DM, Raizman MB, Traboulsi EI. Oral erythromycin treatment for child.
Deviations: The tenderer should clearly read and understand all the terms and conditions, specifications, drawings, etc. mentioned in this tender documents. Deviations, if any, shall be brought out specifically with reference to commercial & technical requirements etc. No deviation shall be allowed from the terms and conditions stipulated in the tender documents and tenders containing deviations are liable to be rejected Deviation, if insisted upon, must be brought out in a separate "Deviation sheet" detailing clause No. of the tender document ; , stipulation as per clause and deviations asked for. Deviation stated elsewhere in the offer except in the deviation sheet shall be treated as NO DEVIATION. Otherwise the tenderer shall be deemed to have accepted all provisions of the Tender documents. Deviations if any, should be along with the cost of with drawl of deviation.
D. J. Capon, and M. A. Martin. 1988. In vitro mutagenesis identifies a region within the envelope gene of the human immunodeficiency virus that is critical for infectivity. J. Virol. 62: 139147. 28. Zennou, V., F. Mammano, S. Paulous, D. Mathez, and F. Clavel. 1998. Loss of viral fitness associated with multiple Gag and Gag-Pol processing defects in human immunodeficiency virus type 1 variants selected for resistance to protease inhibitors in vivo. J. Virol. 72: 33003306. 29. Zhang, Y. M., H. Imamichi, T. Imamichi, H. C. Lane, J. Falloon, M. B. Vasudevachari, and N. P. Salzman. 1997. Drug resistance during indinavir therapy is caused by mutations in the protease gene and in its Gag substrate cleavage sites. J. Virol. 71: 66626670.
Co-administration of indinavir and other drugs that inhibit cyp3a4 may decrease the clearance of indinavir and may result in increased plasma concentrations of indinavir.
Limited data in this patient population, indinavir use is not recommended in HIV-infected pregnant patients see CLINICAL PHARMACOLOGY, Pregnant Patients ; . Antiretroviral Pregnancy Registry To monitor maternal-fetal outcomes of pregnant patients exposed to CRIXIVAN, an Antiretroviral Pregnancy Registry has been established. Physicians are encouraged to register patients by calling 1800-258-4263. Nursing Mothers Studies in lactating rats have demonstrated that indinavir is excreted in milk. Although it is not known whether CRIXIVAN is excreted in human milk, there exists the potential for adverse effects from indinavir in nursing infants. Mothers should be instructed to discontinue nursing if they are receiving CRIXIVAN. This is consistent with the recommendation by the U.S. Public Health Service Centers for Disease Control and Prevention that HIV-infected mothers not breast-feed their infants to avoid risking postnatal transmission of HIV. Pediatric Use The optimal dosing regimen for use of indinavir in pediatric patients has not been established. A dose of 500 mg m2 every eight hours has been studied in uncontrolled studies of 70 children, 3 to 18 years of age. The pharmacokinetic profiles of indinavir at this dose were not comparable to profiles previously observed in adults receiving the recommended dose see CLINICAL PHARMACOLOGY, Pediatric ; . Although viral suppression was observed in some of the 32 children who were followed on this regimen through 24 weeks, a substantially higher rate of nephrolithiasis was reported when compared to adult historical data see WARNINGS, Nephrolithiasis Urolithiasis ; . Physicians considering the use of indinavir in pediatric patients without other protease inhibitor options should be aware of the limited data available in this population and the increased risk of nephrolithiasis. Geriatric Use Clinical studies of CRIXIVAN did not include sufficient numbers of subjects aged 65 and over to determine whether they respond differently from younger subjects. In general, dose selection for an elderly patient should be cautious, reflecting the greater frequency of decreased hepatic, renal or cardiac function and of concomitant disease or other drug therapy. ADVERSE REACTIONS Clinical Trials in Adults Nephrolithiasis urolithiasis, including flank pain with or without hematuria including microscopic hematuria ; , has been reported in approximately 12.4% 301 2429; range across individual trials: 4.7% to 34.4% ; of patients receiving CRIXIVAN at the recommended dose in clinical trials with a median follow-up of 47 weeks range: 1 day to 242 weeks; 2238 patient-years follow-up ; . The cumulative frequency of nephrolithiasis events increases with duration of exposure to CRIXIVAN; however, the risk over time remains relatively constant. Of the patients treated with CRIXIVAN who developed nephrolithiasis urolithiasis in clinical trials during the double-blind phase, 2.8% 7 246 ; were reported to develop hydronephrosis and 4.5% 11 246 ; underwent stent placement. Following the acute episode, 4.9% 12 246 ; of patients discontinued therapy. See WARNINGS and DOSAGE AND ADMINISTRATION, Nephrolithiasis Urolithiasis. ; Asymptomatic hyperbilirubinemia total bilirubin 2.5 mg dL ; , reported predominantly as elevated indirect bilirubin, has occurred in approximately 14% of patients treated with CRIXIVAN. In 1% this was associated with elevations in ALT or AST. Hyperbilirubinemia and nephrolithiasis urolithiasis occurred more frequently at doses exceeding 2.4 g day compared to doses 2.4 g day. Clinical adverse experiences reported in 2% of patients treated with CRIXIVAN alone, CRIXIVAN in combination with zidovudine or zidovudine plus lamivudine, zidovudine alone, or zidovudine plus lamivudine are presented in Table 10 and infliximab.
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Ayloush questioned Taylor's intentions. "By honoring those who have defamed Islam, it can only mean one thing, " he shot back. "Those who seek to demonize our community are doing it by proxy, " he added. Rabbi Haim Beliak, co-director of Jewsonfirst and an exmember of the AJC said that the Congress was not representative of the Jewish community. "This is a renegade organization with an extremely minimal constituency, " he said. "AJC's old LA chapter participated in a study on sweatshops a while back and found out that some had Jewish owners, " the Rabbi added. The findings lead to the closing of the regional office and the formation of a new organization called Progressive Jewish Alliance of which Beliak is a member. InFocus contacted AJC's national office and was told through a.
Chitty, L. S. and D.G. Altman. "New charts for ultrasound dating of pregnancy." Ultrasound in Obstetrics and Gynecology 10: 1997 ; , 174-179, Table 8, 186. Hadlock, F., et al. "Estimating Fetal Age: Computer-Assisted Analysis of Multiple Fetal Growth Parameters." Radiology, 152: 1984 ; , 497-501. Hansmann, M., et al. Ultrasound Diagnosis in Obstetrics and Gynecology. New York: Springer-Verlag, 1986 ; , 431. Osaka University. Ultrasound in Obstetrics and Gynecology. July 20, 1990 ; , 101-102. University of Tokyo, Shinozuka, N. FJSUM, et al. "Standard Values of Ultrasonographic Fetal Biometry." Japanese Journal of Medical Ultrasonics, 23: 12 1996 ; , 886 and intal.
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An uncomfortable feeling in the stomach or belching after eating, also called dyspepsia, or heartburn feeling sick nausea ; , vomiting constipation, diarrhoea headache aching muscles, joints and or bones, which rarely can be severe. flu-like symptoms typically at the start of treatment, such as aching muscles, generally feeling unwell and rarely fever. Swelling of joints Dizziness or spinning sensation Unusual tiredness or weakness Swelling of hands, ankles or feet.
Low plasma concentrations of indinavir are related to virologic treatment failure in hiv-1-infected patients on indinavir-containing triple therapy and invirase.
Canada. Health Canada is warning Canadians of possible drug interactions when the antidepressant trazodone is given in combination with any of the following medications: ketoconazole an antifungal agent ; , ritonavir and indinavir protease inhibitors used in the treatment of HIV ; or carbamazepine an antiepileptic therapy ; . The interactions may affect blood levels of trazodone. If the trazodone blood level increased, patients may experience.
| Indinavir alcoholHomogenized and passed over a 1 ml peptide affinity column containing the peptide zHis-Phe-Phe-Phe-Lys linked via the lysine residue to an agarose resin. Shown is a western blot of column fractions analyzed using polyclonal antibody directed toward the carboxy terminus of rat GLUT4. Lane 1 represents the adipocyte homogenate 5 g ; prior to loading onto the peptide column. Column wash fractions are shown in lanes 2-4 and protein eluted with buffer containing 500 M indinavir is shown in lanes 5-8 50 l lane ; . Rat GLUT4 heterologously expressed in Xenopus oocytes is shown in lane C as a positive control and iressa.
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Homogeneous distribution of human immunodeficiency virus type 1 proviruses within the spleen. Journal of Virology 66, 56425. 26. Korber, B. T. M., Kunstman, K. J., Patterson, B. K., Furtado, M., McEvilly, M. M., Levy, R. et al. 1994 ; . Genetic differences between blood- and brain-derived viral sequences from human immunodeficiency virus type 1-infected patients: evidence of conserved elements in the V3 region of the envelope protein of brain-derived sequences. Journal of Virology 68, 746781. 27. Ball, J. K., Holmes, E. C., Whitwell, H. & Desselberger, U. 1994 ; . Genomic variation of human immunodeficiency virus type 1 HIV-1 ; : molecular analyses of HIV-1 in sequential blood samples and various organs obtained at autopsy. Journal of General Virology 75, 6779. 28. Portegies, P. 1995 ; . HIV-1, the brain, and combination therapy. Lancet 346, 12445. 29. Di Stefano, M., Norkrans, G., Chiodi, F., Hagberg, L., Nielsen, C. & Svennerholm, B. 1993 ; . Zidovudine-resistant variants of HIV1 in brain. Lancet 342, 865. 30. Barry, M., Wild, M., Veal, G., Back, D., Beckenridge, A., Fox, R. et al. 1994 ; . Zidovudine phosphorylation in HIV-infected patients and seronegative volunteers. AIDS 8, F1F5. 31. Stretcher, B. N., Pesce, A. J., Frame, P. T., Greenberg, K. A. & Stein D. S. 1994 ; . Correlates of zidovudine phosphorylation with markers of HIV disease progression and drug toxicity. AIDS 8, 7639. 32. Dianzani, F., Antonelli, G., Turriziani, O., Riva, E., Simeoni, E., Signoretti, C. et al. 1994 ; . Zidovudine induces the expression of cellular resistance affecting its antiviral activity. AIDS Research Human Retroviruses 10, 1478. 33. Back, D. J., Haggard, P. G., Veal, G. J. & Barry, M. G. 1995 ; . Intracellular phosphorylation interactions between nucleoside analogues. In Proceedings of the 5th European Conference on Clinical Aspects and Treatment of HIV Infection, Copenhagen, 1995. Abstract 41, p. 6. 34. Holodniy, M., Katzenstein, D., Mole, L., Winters, M. & Merigan, T. 1996 ; . Human immunodeficiency virus reverse transcriptase Codon 215 mutations diminish virologic response to didanosinezidovudine therapy in subjects with non-syncytium-inducing viral phenotype. Journal of Infectious Diseases 174, 8547. 35. Sylvester, S., Caliendo, A., An, D., Savara, A., Byington, R., Strick, D. et al. 1995 ; . HIV-1 resistance mutations and plasma RNA during ZDV and ddC combination therapy. In Proceedings of the 4th International HIV Drug-resistance Workshop, Sardinia, 1995. Abstract 47. 36. D'Aquila, R. T. 1994 ; . HIV-1 drug resistance: molecular pathogenesis and laboratory monitoring. Clinics in Laboratory Medicine 14, 393422. 37. Smith, M. S., Koerber, K. L. & Pagano, J. S. 1994 ; . Long-term persistence of zidovudine resistance mutations in plasma isolates of human immunodeficiency virus type 1 of dideoxyinosine-treated patients removed from zidovudine therapy. Journal of Infectious Diseases 169, 1848. 38. Moyle, G. J. 1996 ; . Activity and role of lamivudine in the treatment of adults with human immunodeficiency virus type 1 infection: a review. Expert Opinion on Investigational Drugs 5, 91324. 39. Havlir, D., Cheeseman, S. H., McLaughlin, M., Murphy, R., Erice, A., Spector, S. A. et al. 1995 ; . High-dose nevirapine: safety, pharmacokinetics and antiviral effect in patients with human immunodeficiency virus infection. Journal of Infectious Diseases 171, 53745. 40. Japour, A. J., Welles, S., D'Aquila, R. T., Johnson, V. A., Richman, D. D., Coombs, R. W. et al. 1995 ; . Prevalence and clinical significance of zidovudine resistance mutations in human immunodeficiency virus isolated from patients after long-term zidovudine treatment. Journal of Infectious Diseases 171, 11729. 41. D'Aquila, R. T., Johnson, V. A., Welles, S. L., Japour, W. A., Kuritzkes, D. R., DeGruttola, V. et al. 1995 ; . Zidovudine resistance and HIV-1 disease progression during antiretroviral therapy. Annals of Internal Medicine 122, 4018. 42. Havlir, D. V., Eastman, S., Gamst, A. & Richman, D. D. 1996 ; . Nevaripine-resistant human immunodeficiency virus: Kinetics of replication and estimated prevalence in untreated patients. Journal of Virology 70, 78949. 43. Larder, B. A., Darby, G. & Richman, D. D. 1989 ; . HIV with reduced sensitivity to zidovudine AZT ; isolated during prolonged therapy. Science 243, 17314. 44. Boucher, C. A. B., Tersmette, M., Lange, J. M. A., Kellam, P., de Goede, R. E., Mulder, J. W. et al. 1990 ; . Zidovudine sensitivity of human immunodeficiency viruses from high-risk, symptom-free individuals during therapy. Lancet 336, 58590. 45. Lopez-Galindez, C., Rojas, J. M., Najera, R., Richman, D. D. & Perucho, M. 1991 ; . Characterization of genetic variation and 3 azido-3 -deoxythymidine resistance mutations of HIV by the RNase mismatch cleavage method. Proceedings of the National Academy of Sciences of the USA 88, 42804. 46. Larder, B. A., Kellam, P. & Kemp, S. D. 1991 ; . Zidovudine resistance predicted by direct detection of mutants in DNA from HIV-infected lymphocytes. AIDS 5, 13744. 47. De Jong, M. D., Veenstra, J., Stilianakis, N. I., Schuurman, R., Lange, J. M. A., de Boer, R. J. et al. 1996 ; . Host-parasite dynamics and outgrowth of virus containing a single K7OR amino acid change in reverse transcriptase are responsible for loss of human immunodeficiency virus type 1 RNA load suppression by zidovudine. Proceedings of the National Academy of Sciencies of the USA 93, 95016. 48. Mayers, D. L., McCutchan, F. E., Sanders-Buell, E. E., Merritt, L. I., Dilworth, S., Fowler, A. K. et al. 1992 ; . Characterization of HIV isolates arising after prolonged zidovudine therapy. Journal of Acquired Immune Deficiency Syndrome 5, 74959. 49. St Clair, M. H., Martin, J. L., Tudor-Williams, G., Bach, M. C., Vavro, C. L., King, D. M. et al. 1991 ; . Resistance to ddI and sensitivity to AZT induced by a mutation in HIV-1 reverse transcriptase. Science 253, 15579. 50. Larder, B. A. 1992 ; . 3 Azido-3 deoxythymidine resistance suppressed by a mutation conferring human immunodeficiency virus type 1 resistance to nonnucleoside reverse transcriptase inhibitors. Antimicrobial Agents and Chemotherapy 36, 26649. 51. Boucher, C. A. B., Cammack, N., Schipper, P., Schuurman, R., Rouse, P., Wainberg, M. A. et al. 1993 ; . High-level resistance to ; enantiomeric 2 deoxy-3 -thiacytidine 3TC ; in vitro is due to one amino acid substitution in the catalytic site of HIV-1 reverse transcriptase. Antimicrobial Agents and Chemotherapy 37, 22314. 52. Tisdale, M., Kemp, S. D., Parry, N. R. & Larder, B. A. 1993 ; . Rapid in vitro selection of human immunodeficiency virus type 1 resistant to 3 -thiacytidine inhibitors due to a mutation in the YMDD region of reverse transcriptase. Proceedings of the National Academy of Sciences of the USA 90, 56536.
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