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BULLS FOR SALE--Charolais, Simmental, Red and Black, Red Angus, X Simmental. Yearlings and two year olds. LVV Ranch 780 ; 582-2254. --453-9J21c 3RD MIDWESTERN HORNED Hereford Sale--Thursday, Feb. 7, 2002, 1: 00 p.m. M.S.T. Lloydminster Exhibition, Lloydminster, SK AB. 42 two year old bulls - semen tested; 5 bull calves; 30 bred commercial heifers, 15 bred registered heifers - preg. checked; 4 registered heifer calves; 10 commercial heifer calves. Call for a catalogue. Delivery available. Come early for dinner! Rob Bannerman - 1-306-845-2764, Lanni Bristow - 1-780-943-2236, David Mitchell - 1-306-893-2838, Mike Newman - 1-306-825-2701. --959-16J3c. Lance Chilton, MD Over a number of years of American Academy of Pediatrics involvement, I've written two or three resolutions, trying to affect Academy function or advocacy. I feel strongly that the AAP continues to be a very effective organization, largely because it looks after the needs of children first and is known for sticking its collective head out for our patients. So every once in a while I have tried to push it one way or another. I know that Academy resolutions take a long time to get through it's quite a process, and that, even when passed at the end of that process, they are only advisory to the AAP Board. I also know that each one is taken seriously, and that the author will get a considered response on each one of them that is passed. There's never a time when one can't begin writing a resolution, and there's plenty of help available for those who need it. I did need it with my first; now I can offer help to anyone who needs it. You write a resolution in a certain format, available on the Member's Channel at : aap moc displaytemp resolution template 08.doc. You write some "Whereases", indicating the premises on which you're asking the Academy to act. Then you write one or two, "Resolveds", stating clearly the action you want the Academy to take. The resolution is usually then considered by the chapter NMPS, in our case ; and then by the AAP district we're in District VIII ; . It is shaped and coddled along the way, and then numbered and submitted to close scrutiny at the Annual Leadership Forum near Chicago. There, all the presidents and vice-presidents from the country's 60 + chapters vote on each. President Rob Miller and Vice-President Joanne Ray were part of the group this past March. All passed resolutions then go to the AAP Board and are parceled out to various Academy entities, usually committees and sections, for comment and usually for action. Usually the "Whereases" disappear during this process, so the "Resolveds" have to stand on their own. The resolutions voted in the "Top Ten" are given special consideration. My 2007 ALF resolution, "RESOLVED, that the Academy publicize the importance of and the availability of goods produced and distributed using fair trade principles, through educational means such as discussion at the NCE and an article or articles in AAP News, and further be it RESOLVED, that wherever possible the Academy purchase goods produced and distributed through organizations adhering to fair trade principles." was passed by the ALF in March, and I heard nothing about it for the next four months. I wasn't surprised; aside from a few very urgent resolutions, it takes a while to get answers through the volunteers and staff at the AAP. Then at Portland's District Meeting in September, Academy President Jay Berkelhamer asked me what I thought about the.

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Malignant pleural mesothelioma: results in 183 patients. J Thorac Cardiovasc Surg 1999; 117: 54 Rusch VW, Rosenzweig K, Venkatraman E, et al. A phase II trial of surgical resection and adjuvant high-dose hemithoracic radiation for malignant pleural mesothelioma. J Thorac Cardiovasc Surg 2001; 122: 788 Vogelzang NJ, Rusthoven J, Symanowski J, et al. A phase III study of pemetrexed in combination with cisplatin versus cisplatin alone in patients with malignant pleural mesothelioma. J Clin Oncol 2003; 21: 2636 Martinez-Moragon E, Aparico J, Rogado MC, et al. Pleu rodesis in malignant pleural effusions: a randomized study of tetracycline versus bleomycin. Eur Respir J 1997; 10: 2380 Yim APC, Chan ATC, Lee TW, et al. Thoracoscopic talc insufflation versus talc slurry for symptomatic malignant pleural effusion. Ann Thorac Surg 1996; 62: 16551658 Zellos LS, Sugarbaker DJ. Multimodality treatment of diffuse malignant pleural mesothelioma. Semin Oncol 2002; 29: 4150 Rusch V, Saltz L, Venkatraman E, et al. A phase II trial of pleurectomy decortication followed by intrapleural and systemic chemotherapy for malignant pleural mesothelioma. J Clin Oncol 1994; 12: 1156 Rusch VW. Pleurectomy decortication in the setting of multimodality treatment for diffuse malignant pleural mesothelioma. Semin Thorac Cardiovasc Surg 1997; 9: 367372 Rice TW, Adelstein DJ, Kirby TJ, et al. Aggressive multimodality therapy for malignant pleural mesothelioma. Ann Thorac Surg 1994; 58: 24 Baas P. Chemotherapy for malignant mesothelioma: from doxorubicin to vinorelbine. Semin Oncol 2002; 29: 62 Lerner HJ, Schoenfeld DA, Martin A, et al. Malignant mesothelioma: the Eastern Cooperative Oncology Group ECOG ; experience. Cancer 1983; 52: 19811985 Magri MD, Veronesi A, Foladore S, et al. Epirubicin in the treatment of malignant mesothelioma: a phase II cooperative study. Tumori 1991; 77: 49 Bajorin D, Kelsen D, Mintzer DM. Phase II trial of mitomycin in malignant mesothelioma. Cancer Treat Rep 1987; 71: 857 Zidar BL, Green S, Pierce HL, et al. A phase II evaluation of cisplatin in unresectable diffuse malignant mesothelioma: a Southwest Oncology Group Study. Invest New Drugs 1988; 6: 223226 Planting AST, Schellens JHM, Goey SH, et al. Weekly high-dose cisplatin in malignant pleural mesothelioma. Ann Oncol 1994; 5: 373374 Raghavan D, Gianoutsos P, Bishop J, et al. Phase II trial of carboplatin in the management of malignant mesothelioma. J Clin Oncol 1990; 8: 151154 Harvey VJ, Slevin ML, Ponder BA, et al. Chemotherapy of diffuse malignant mesothelioma: phase II trials of singleagent 5-fluorouracil and adriamycin. Cancer 1984; 54: 961 Solheim OP, Saeter G, Finnanger AM, et al. High-dose methotrexate in the treatment of malignant mesothelioma of the pleura: a phase II study. Br J Cancer 1992; 65: 956 van Meerbeeck J, Debruyne C, van Zandwijk N, et al. Paclitaxel for malignant pleural mesothelioma: a phase II study of the EORTC Lung Cancer Cooperative Group. Br J Cancer 1996; 74: 961963 Vorobiof DA, Rapoport BL, Chasen MR, et al. Malignant pleural mesothelioma: a phase II trial with docetaxel. Ann Oncol 2002; 13: 412 Steele JPC, Shamash J, Evans MT, et al. Phase II study of vinorelbine in patients with malignant pleural mesotheli.

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1. Roche, Y., Fay, M. & Gougerot-Pocidalo, M. A. 1987 ; . Effects of quinolones on interleukin 1 production in vitro by human monocytes. Immunopharmacology 13, 99109. 2. Bailly, S., Mahe, Y., Ferrua, B., Fay, M., Tursz, T., Wakasugi, H. et al. 1990 ; . Quinolone-induced differential modification of IL-1 and IL-1 production by LPS-stimulated human monocytes. Cellular Immunology 128, 27788. 3. Riesbeck, K. & Forsgren, A. 1990 ; . Selective enhancement of synthesis of interleukin-2 in lymphocytes in the presence of ciprofloxacin. European Journal of Clinical Microbiology and Infectious Diseases 9, 40913. 4. Imada, T., Miyazaki, S., Nishida, M., Yamaguchi, K. & Goto, S. 1992 ; . In vitro and in vivo antibacterial activities of a new quinolone, OPC-17116. Antimicrobial Agents and Chemotherapy 36, 5739. 5. Kita, M., Ohmoto, Y., Hirai, Y., Yamaguchi, N. & Imanishi, J. 1992 ; . Induction of cytokines in human peripheral blood mononuclear cells by mycoplasmas. Microbiology and Immunology 36, 50716. 10 Verbraecken J, Van de Heyning P, De Backer W et al. Body surface area in normal-weight, overweight, and obese adults. A comparison study. Metabolism 2006; 55: 515524. Egorin MJ, Van Echo DA, Olman EA et al. Prospective validation of a pharmacologically based dosing scheme for the cis-diamminedichloroplatinum II ; analogue Cancer Res 1985; 45: 6502 Alberts DS, Dorr RT. New perspectives on an old friend: Optimizing carboplatin for the treatment of solid tumors. The Oncologist 1998; 3: 1534. Lichtman SM, Skirvin JA, Vemulapalli S. Pharmacology of antineoplastic agents in older cancer patients. Crit Rev Oncol Hematol 2003; 46: 101114. Wilkinson GR. Drug metabolism and variability among patients in drug response. N Engl J Med 2005; 352: 22112221. Scripture CD, Figg WD. Drug interactions in cancer therapy. Nat Rev Cancer 2006; 6: 546 Felici A, Verweij J, Sparreboom A. Dosing strategies for anticancer drugs: The good, the bad and body-surface area. Eur J Cancer 2002; 38: 1677 Ratain MJ, Mick R, Schilsky RL et al. Pharmacologically based dosing of etoposide: A means of safely increasing dose intensity. J Clin Oncol 1991; 9: 1480 De Jongh FE, Verweij J, Loos WJ et al. Body-surface area-based dosing does not increase accuracy of predicting cisplatin exposure. J Clin Oncol 2001; 19: 37333739. Baker SD, Verweij J, Rowinsky EK et al. Role of body surface area in dosing of investigational anticancer agents in adults, 19912001. J Natl Cancer Inst 2002; 94: 18831888. Rudek MA, Sparreboom A, Garrett-Mayer ES et al. Factors affecting pharmacokinetic variability following doxorubicin and docetaxel-based therapy. Eur J Cancer 2004; 40: 1170 Grochow LB, Baraldi C, Noe D. Is dose normalization to weight or body surface area useful in adults? J Natl Cancer Inst 1990; 82: 323325. Nguyen L, Chatelut E, Chevreau C et al. Population pharmacokinetics of total and unbound etoposide. Cancer Chemother Pharmacol 1998; 41: 125 Cosolo WC, Morgan DJ, Seeman E et al. Lean body mass, body surface area and epirubicin kinetics. Anticancer Drugs 1994; 5: 293297. Gurney HP, Ackland S, Gebski V et al. Factors affecting epirubicin pharmacokinetics and toxicity: Evidence against using body-surface area for dose calculation. J Clin Oncol 1998; 16: 2299 Dobbs NA, Twelves CJ. What is the effect of adjusting epirubicin doses for body surface area? Br J Cancer 1998; 78: 662 Ralph LD, Thomson AH, Dobbs NA et al. A population model of epirubicin pharmacokinetics and application to dosage guidelines. Cancer Chemother Pharmacol 2003; 52: 34 Loos WJ, Gelderblom H, Sparreboom A et al. Inter- and intrapatient variability in oral topotecan pharmacokinetics: Implications for body-surface area dosage regimens. Clin Cancer Res 2000; 6: 26852689. Mathijssen RH, Verweij J, de Jonge MJ et al. Impact of body-size measures and eplerenone.

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Table 2. Combination chemotherapy in thymic carcinoma * Regimen PE + epirubicin: cisplatin, etoposide, epirubicin PE + bleomycin: cisplatin, etoposide, bleomycin No. of patients 3 Response 3 Reference Macchiarini et al. 11 and epogen. How Abortion is Provided Abortion ends a pregnancy before birth takes place. When an embryo or fetus dies in the uterus and is expelled by the body, it is called a spontaneous abortion. After 20 weeks of pregnancy a spontaneous abortion is also called a "miscarriage." When a woman decides to end her pregnancy voluntary, she has an induced abortion. Approximately 88 percent of all induced abortions are performed during the first trimester - the first three months of pregnancy. In fact, more than half are preformed within the first two months of pregnancy CDC, 2002 ; . These abortions are usually performed at a clinic, health center, or in a doctor's office, and women are usually able to return home an hour or so later. Medical Abortion In some cases, women can choose to use a combination of drugs to end their pregnancies. This is called medical abortion. Medical abortion does not require surgery. The medications used are prescribed by a clinician. A small percentage of medical abortions fail. In these cases, surgical procedures are required to end the pregnancy. Medical abortion is not available from all abortions providers. Follow-up visits are important for women seeking medical abortions. Candidates for medical abortion must be able to complete the regimen of drugs and to make between one and three visits to the medical provider. Two combinations of medications can be used for medical abortion.
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And they laid hands on her, and she went the way that the horses of the kings went out and was slain there. And Jehoiada made a bond both between the Lord and the king, and between the people and the Lord, that they should be the Lords people: and also between the king and the people. Then all the people of the land went into the house of Baal, and destroyed his altars, and brake down his Images lustily, and slew Nathan the priest of Baal before the altar. And the priest set watchmen in the house of the Lord, and took the rulers over hundreds and the captains and the guard and all the people of the land: And they brought the king from the house of the Lord and went the way of the gate of the guard of the kings house. And he sat him down on the seat of the kings. And all the people of the Lord rejoiced, and the city was in quiet. And they slew Athaliah with the sword in the house of the king. [Chpt 12] Jehoas was seven years old when he was made king. And he began to reign the seventh year of Jehu, and reigned forty year in Jerusalem. His mothers name was Zebiah Bersabe. And he did that pleased the Lord, as long as Jehoiada the Priest informed him. But they took not away the hill altars, for the people slew and offered still in the hill altars. And Jehoas said to the priest: all the silver that is dedicate and brought to the house of the Lord in current money, that is to say, the money that every man is set at, with all the money that every mans heart giveth him to bring into the house of the Lord, let the priests take it to them, every man of his acquaintance, and let them repair the broken places of the temple in all the places where ought is found decayed. Neverthelater the priests had not mended unto the twenty third year of Jehoas, that was decayed in the temple. Then and epoprostenol.

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614 two drugs. The feasibility of the combination of gemcita- Table 1. Characteristics of patients. bine and epirubicin has already been tested in advanced Number of patients breast cancer in chemotherapy naive patients [15]. Age On these grounds, in 1997 we started a dose-finding Median study with escalated doses of epirubicin starting dose Range 60 mg m 2 , day 1 ; and gemcitabine starting dose 800 Cell type Serous mg m 2 , day 1 and day 8 ; for the treatment of advanced Mucinous platinum-refractory or resistant ovarian cancer. Bucil 27 ; . D ; Any combination COP, CHOP, or chlorambucil [Chl] plus epirubicin [Epi] ; versus chlorambucil. NS not statistically significant; SD standard deviation; and logrank logrank test. Deaths and person-years refer to the 1-year interval under which they are printed except for the final period ; . P values are two-sided and eprosartan.

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French Adjuvant Study Group. Benefit of a high-dose epirubicin regimen in adjuvant chemotherapy for node-positive breast cancer patients with poor prognostic factors: 5-year follow-up results of French adjuvant study group 05 randomized trial. J Clin Oncol 2001; 19: 602611.

R, Thomas RV, Graham J, et al. administration of 3'-azido-2'3'to patients with AIDS and erbitux. Clarification of the juice and the early stages of fermentation, these observations suggest that in seasons with rain close to harvest, powdery mildew may predispose the grapes to bunch rot and contamination by spoilage organisms. The Chardonnay grapes with 30% infection gave juice and wine a chemical composition quite different from the lower infection levels and the disease-free control. Severely affected grapes had lower total soluble solids and the corresponding wine had less alcohol, indicating delayed grape maturity. The concentrations of `total phenolics'.

VSUP was performed with a 7.5 MHz probe Sonoline SI-45; Siemens, Erlangen, Germany ; . The probe was placed in a position to provide a sagittal section of the whole uterus and was maintained in a fixed position over a period of 5 min. The whole scan was videotaped for quantitative assessment of uterine peristalsis, the tape being replayed at 5 regular speed in order to estimate precisely the frequency of the contraction waves. This also facilitated determination of the wave direction cervicofundal versus fundocervical peristalsis and ergotamine. By Dr. Karen Gelmon, MD, FRCPC Chair, Breast Tumour Group, BC Cancer Agency strategy of how to treat a postmenopausal women with a estrogen sensitive tumour, whether it is best to treat with Arimidex for five years, or tamoxifen for five years followed by Femara for five years, or tamoxifen for 3 years followed by Aromasin for 3 years? They do provide further evidence that there is a survival benefit potential and that they are safe and well tolerated. Women should have a discussion of these options with their oncologist. As well, there was an update of a previously presented trial of postmenopausal women with estrogen receptor positive tumours with recurrent disease who were treated with exemestane or tamoxifen again showing a beneficial effect for the newer drug. There were further presentations on the roles of bisphosphanates prevents spread of cancer into bone ; and on trastuzumab Herceptin ; . In one presentation, using trastuzumab in the neoadjuvant setting treatment given prior to surgery ; , the researchers bravely gave the drug concurrently with epirubicin chemotherapy drug ; despite concerns about the risk of heart failure. They showed very impressive results in terms of the number of women whose tumours had decreased prior to surgery and did not show significant early side effects on the heart. However, with fewer than two dozen women and without long term follow-up, it is not yet clear how safe this protocol will be and at this time it should be further studied. Studies in bisphosphanates confirmed their safety and efficacy and we need to await the results of the recently closed study of clodronate first generation bisphosphanate ; in the adjuvant setting B34 from the NSABP ; to see if they should be more broadly instituted. In one session focusing on women with genetic mutations which increase the risk of breast cancer, there was further emphasis placed on the role of MRI screening of the breast over mammography alone. Although MRI has been shown to have very good sensitivity, the specificity is poor, meaning it shows and epirubicin.

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John, and Bessie Liddell, married in Dunbar - 1567 John, hammerman, and Margaret Ormestoun, mar. in the Kirk of Halyroodhous be Mr. George Leslie p. 26 Oct. m. Fryday, 19 Dec. 1651 John, cordiner, and Marie Gray, mar. in the Kirk of Halyroodhous by Mr John Paterson, minister at the Tron Kirk of Edinburgh p. 24 Oct., m. Thursday, 25 Nov. 1669 John, in this Congregation, and Isobell Straton in Edinburgh, mar. within the Kirk of Halyroodhous be Mr. James Nairne, minister p. 27 Mar., m. Fryday, 29 April 1659 John, in North Leith, and Jean Slush p. 14 Aug., mar. at North Leith, 8 Sept. 1670 John, servant to the Earl of Murray, and Margaret Craufurd, servant to Colin M'Kenzie, goldsmith in Edinburgh 21 Oct. 1707 John, harnishmaker, and Margaret Halden, servitrix to James Brotherstains, late conveener of the Trades 22 May 1722 John, coachmaker, and Barbra Smith, daughter of Robert Smith 30 Dec. 1785 John, wright, residenter, and Euphan Law, daughter of James Law, wright in Drem ; witness, Andrew Paterson 4 April 1795 John, mason, and Margaret Lawson, daughter of Mr. William Lawson, shurgeon in Leith 1 Aug. 1798 Livingston, and James Mackie, labourer 20 May 1781 Margaret, in Edinburgh, and George Hay Sabbath, 30 Oct. 1653 Margaret, and James Cargill 26 Jan. 1690 Margaret, and Patrick Gray, brewar, mar. in the Church of Halyroodhous by Mr. James Kid, minister p. 17 Sept., m. Tuysday, 10 Oct. 1671 Margaret, and John Stevinsone, barbour, mar. in the Kirk of Cannongate be Mr. George Leslie p. 7 May, m. Tuysday, 27 June 1654 Margaret, ane of Edinburgh, and Jon Willsone, mar. at Edinburgh 25 Jan. 1648 Margaret, relict of the deceast Mr. James Stewart, schoolmaster at Prestonhall, and Daniel Nasmith, sadler 4 July 1709 Margaret, and Mungo M'Onst, journeyman tailor 23 Aug. 1745 Margaret, and John Brown 6 Dec. 1745 Margaret, daughter to the deceased Hugh Paterson, weaver of the parish of Ceres, and Henry Jamison, journeyman wright 20 Dec. 1773 Margaret, daughter of the deceased James Paterson, labourer, and Thomas Rillie, stockingmaker 12 July 1786 Margaret, daughter of John Patterson, labourer, and Robert Johnston, tailor in Dunbar 2 Jan. 1792 Margaret, daughter of John Paterson, wright at Eymouth, and Peter Hendry, tinnsmith 28 Nov. 1795 Margaret, daughter of James Paterson, tailor, and Thomas Main, gardener in the Abbey 23 Feb. 1796 Marie, and James Crookshanks, weaver in Tranent, mar. be Mr. James Kid, minister p. 12 April, m. Tuesday, 18 June 1672 Marion, indueller, and Alexander Thomson, servant to the Earle of Leven 4 July 1727 Marion, in the parish of the West Kirk, and William Willson, servant to Mr. Knox, tacksman, in the Kings Park 18 July 1755 Mary, daughter laufull to George Patersone, late fermerer in Wright Houss, and William Frazer, Saidler, burges and freeman p. 17 July, m. 12 Aug. 1697 and erlotinib.

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Test scored according to: -Number correct answers -Median time for correct answers sec ; -Score -Number of picture viewings -Median time for incorrect answers sec ; -Working time The following norms are available: S1: a sample of 163 healthy adults S2: a representative sample from a Population of 677 people Drivers with increased risk- a cohort sample of 4104 from traffic psychology research in Germany and Austria. See below.
Arm, respectively, and grade IV infection or febrile neutropenia in 8% and 0.4% of the patients. The frequency of grade III--IV thrombocytopenia was 5.5% vs. 2% in FEC 100 and FEC 50, with haemorrhagic complications grade III--IV ; rarely observed 1 patient in FEC 100 ; . Two patients in each arm or 1 and 0.8% ; died of infectious complications related to neutropenia. The incidence of grade III--IV nausea and vomiting was comparable in the two treatment arms 30% and 26% ; , but patients receiving the high dose experienced more mucositis, with grade III--IV in 10% of those treated with FEC 100 or 3% of the cycles ; vs. 0.4% of those receiving FEC 50 or 0.1% of the cycles ; . Alopecia was seen in almost all patients, with no difference between the groups. The number of patients who experienced a 15% absolute value ; LVEF drop as compared to baseline or 10% LVEF drop below the lower normal limit was 23 and 12 in FEC 100 and FEC 50, respectively; of these, 7 and 4 cases were taken off study. The median cumulative epirubicin dose was 522 mg m2 range 83-926 mg m2 ; and 289 mg m2 range 43-492 mg m2 ; in FEC 100 and FEC 50, respectively. The frequency of cardiac events leading to therapy discontinuation was not different in the two groups 10 patients or 5% vs. 8 patients or 3% in FEC 100 and FEC 50, respectively ; . Two patients experienced CHF during treatment one in each arm, after two and one cycle, respectively ; , one had a mitral valve prolapse, but none had received either prior anthracyclines or radiotherapy to the mediastinum. Both patients recovered. Of the remaining patients 11 had a LVEF drop 7 in FEC 100 ; , 4 had ECG alterations 1 in FEC 100 ; and one in the FEC 100 group had asymptomatic cardiomegaly. Another patient treated with FEC 100 was reported to have mild signs and symptoms of CHF after treatment completion. Deaths on therapy were 7 3% ; in FEC 100 arm and 3 1% ; in FEC 50. The causes of death were progressive disease 4 vs. 1 in high vs. conventional dose, respectively ; , toxicity related to myelosuppression 2 in each arm ; and a cerebro-vascular accident in the absence of thrombocitopenia one case in FEC 100 ; . Compliance in the completion of the quality-of-life questionnaire was very poor, with approximately half of and ertapenem.
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