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Requisite for the admission to a stressful environment, such as the Military Academy, was performed by a team of board-qualified psychiatrists. It is well known that for various reasons, individuals undergoing a psychological assessment may over-report or underreport problems. Moreover, as the stakes involved in a psychological evaluation increase, so does the likelihood that the individual will distort his or her responses Sellbom et al. 2005 ; . Hence, the military academy of Pozzuoli, Italy, requires every candidate to be evaluated by self-administering a standardized personality test, the Minnesota Multiphasic Personality Inventory-2 MMPI-2; Sellbom et al. 2005 ; , followed by a brief colloquium with the team of boardqualified psychiatrists, in order to confirm the orientation given by the test. According to the psychiatric evaluation each subject was judged either as potentially either successful S ; , or not-successful NS ; in managing interpersonal relationships. Statistical analysis was performed by using EpiInfoTM 3.3. Differences among classes were calculated by the 2 test. Linear trends in proportion were tested using 2 test for trend Mantel, 1963 4. Leading Brands on the Market 4.1 Overview 4.2 Leading Brands Revenue & Market Share 4.3 Profiles of Leading Brands 4.3.1 Neulasta Peg-Filgrastim ; 4.3.2 Neupogen Filgrastim ; 4.3.3 Neutrogin Granocyte Lenograstim.
P119 Interim Results of a Phase II Study of Ancestim SCF ; and Twice-Daily High-Dose Filgrastim for Mobilization of Peripheral Blood Stem Cells PBSC ; in Patients with Indolent Lymphoproliferative Disorders Previously Treated With Fludarabine Kirsten Herbert, Susan Morgan, John Reynolds, Henry Januszewicz, Miles Prince, David Westerman, Max Wolf, John Seymour Peter MacCallum Cancer Centre, East Melbourne VIC Indolent histology and prior fludarabine exposure are risk factors for impaired PBSC mobilization. In our previous analysis of patients with prior fludarabine treatment Leukemia May 2004 ; only 14 of 41 35% ; achieved PBSC yields of 2x106 kg using growth factor + - chemotherapy. Aim: To assess mobilization efficacy of an alternative approach using ancestim plus high-dose filgrastim without chemotherapy, given the known synergy between G-CSF and SCF. Method: Study drugs: Ancestim SCF ; 20 g kg day sc from day 1; Filgrastim 12g kg BD sc from day 4. Apheresis scheduled to commence day 6, provided PB CD34 + count ~3 x 106 L. Patients from the previous study served as historical controls. Result: Thus far, of 16 patients recruited, 14 are evaluable; median age 51 years range 31-63 ; , median cumulative fludarabine dose 663 480-900 ; g. Nine of 14 64% ; of patients collected 2 x 106 kg CD34 + cells, compared to 35% with the previous approach p 0.06 ; . The median CD34 + yield was 2.21 lower and upper quartiles 1.61-3.26 ; x 106 kg compared to 1.93 0.2-2.64 ; x106 kg in the previous study. Means were not significantly different. Mobilization scheduling was predictable: all patients commenced apheresis on day 6 n 9 ; The regimen was well tolerated, with injection site reactions [grade 1-2 n 8 ; ], bone pain [grade 1-2 n 8 ; ], lethargy grade 1-2 n 3 ; ], and no serious adverse events or infectious complications. Conclusion: The regimen of ancestim and BD high-dose filgrastim shows promising efficacy in this interim analysis of patients previously treated with fludarabine with good tolerance, predictable kinetics and no infectious morbidity. P120 Infectious Complications of Fludarabine Therapy in Low-Grade Lymphoproliferative Disorders Christopher Ward, Lois Jacklin, Chris Arthur, Luke Coyle, Keith Fay Department of Haematology and Transfusion Medicine, Royal North Shore Hospital, St Leonards, NSW Fludarabine is a potent nucleoside analogue with activity against lymphoid and myeloid malignancies. Therapy with fludarabine-containing regimens can be associated with major infectious complications, due to prolonged immunosuppression. The optimal regimen for prophylaxis of infections has not been established. Aim: A retrospective review of 16 patients with low-grade lymphoproliferative disorders CLL or NHL ; treated with fludarabine was performed in our institution to assess the safety of this agent and the efficacy of infection prophylaxis. Result: Twelve of 16 patients 75% ; were admitted on at least one occasion for probable infection following therapy. Increased age was associated with an increased number of infections. All of the patients admitted were neutropenic and the respiratory tract was the most common site of infection. Fifty percent of these infections were attributed to bacterial causes E.coli in 3 cases ; . Viral infections were predominantly due to reactivation of herpes viruses and 75% of fungal infections were due to Candida spp. Eighty-three percent of the patients developing infections after fludarabine had also been admitted after previous therapies. All patients received some form of infection prophylaxis; 15 16 patients were on cotrimoxazole and no cases of Pneumocystis carinii pneumonia PCP ; were seen. 50 percent of patients who developed viral infections were receiving valacyclovir prophylaxis. All 4 patients who experienced fungal infections had been treated with fluconazole. Conclusion: Infectious complications are common after fludarabine therapy in this group of patients; PCP prophylaxis is effective, but improved strategies are needed to prevent bacterial, viral and fungal infections.

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Blood cell WBC ; count, platelets and haemoglobin were analysed. On the basis of the WBC and platelet cell counts, the doses were adjusted aiming at an equivalent WBC and platelet toxicity for each of the following courses Table 2 ; . Patients who were randomly assigned to the autologous marrowsupportive high-dose chemotherapy arm were treated with three courses of F600E60C600 mg m2 ; , if for administrative reasons the marrow-supported high-dose therapy could not be delivered within 34 weeks after the third course, a fourth course of standard F600E60C600 mg m2 ; was recommended. To facilitate harvest of peripheral blood stem cells, the cyclophosphamide dose at the third FEC course was escalated to 1200 mg m2 together with filgrastim at 5 lg kg. CTCb 6000 mg m2 cyclophosphamide, 500 mg m2 thiotepa and 800 mg m2 carboplatin ; was given as intravenous infusion over 4 days on days 7 to 4 and the stored autologous peripheral stem cells with a recommended dose of at least 2 106 CD34 + cells kg were transfused on day 0. These patients were started on filgrastim at 5 lg kg, the day after marrow transfusion. All randomised patients received locoregional radiotherapy in 2 Gy fractions, 5 fractions week, to a total dose of 4650 Gy. The target volumes were mastectomy scar area or breast parenchyma, axillary-, supraclavicular- infraclavicular- and parasternal lymph nodes. The latter lymph nodes were only included if considered to be technical feasible or not resulting in too high doses into the myocardium. All patients were given tamoxifen at 20 mg per day for 5 years nonconcurrently with the delivered chemotherapy and irrespective of hormone receptor status. Gout and gouty is also plasma arthritis indicated levels.

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Study design Data was reviewed from 153 consecutive patients with untreated aggressive B-cell lymphoma enrolled on DA-EPOCH-based protocols at the National Cancer Institute between May 1993 and August 2002. Histologies included diffuse large B-cell lymphoma, Burkitt's lymphoma and mantle cell lymphoma. Patients with human immunodeficiency virus infection were included. To control for confounding causes of neutropenia and ensure adequate followup, the analysis was restricted to 130 patients in complete remission who had hematopoietic recovery with an absolute neutrophil count 1000 mm3 after treatment and were observed for at least 12 months. Patients had no other identifiable causes of neutropenia and had no recent changes to medications before or during the LON. Patients were routinely evaluated with complete blood counts and computerized tomography scans every three months for the first year. LON was defined as a neutrophil count 500 cells mm3 occurring at least 60 days after the last treatment. All patients received DA-EPOCH-based treatment: [Etoposide 50 mg m2 day CIV days 1-4; Prednisone 60 mg m2 BID PO days 1-5; Oncovin vincristine ; 0.4 mg m2 day CIV days 1-4; Cyclophosphamide 750 mg m2 IV day 5 and Hydroxydaunorubicin doxorubicin ; 10 mg m2 day CIV days 1-4]. Filgrastim was administered from day 6 until neutrophil recovery. Drug doses were adjusted as previously described and treatment was administered for 3 to 8 cycles and flax.

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Georg Philipp Telemann Magdeburg, 1681 - Hamburg, 1767 ; is among the most prolific German composers of the 18th cen tu ry. His output -- which numbers almost 6, 000 works of which many are unfortunately lost -- consists of 12 series of cantatas for the 52 Sundays of the year, about 100 oratorios, 44 passions, more than 600 French overtures, 40 operas and hundreds of concerti, quartets and sonatas. This spectacular productivity can be explained in part by the composer's long life; born four years before Bach at the height of the baroque, he died aged 86, just as Haydn was establishing the guidelines of the classical period. Tel em a n who was a child prod i gy, received his first music lessons early in his life. His parents were de s cendants of a host of Lutheran pastors and preferred, however, that he pursue a brilliant university career. Even though he learned the vi o l lute and harpsichord, he remained hostile to the idea of com. Stem cell mobilisation and collection Consult local chemotherapy transplant protocols for details of drug administration ; Cyclophosphamide G-CSF Day 1 Day 0 start allopurinol, ciprofloxacin, fluconazole, chlorhexidine mouthwash insert central line Cyclophosphamide 3g m2 iv over 3-4 hours Mesna 1.6g m2 15 mins prior cyclophosphamide infusion, then same dose at 3, 6, 9, hours post cyclophosphamide. Day + 1 start filgrastim 10g kg day subcutaneously PBSC usually harvested between day + 11, 12 post cyclophosphamide, when CD34 + count 1x109 l. Daily FBC from day + 8 is required. Threshold of 2.0 x106 kg CD34 + cells should be obtained. High dose therapy should commence between day13 and 28 post cyclophosphamide. Dose adjustment for impaired renal function Creatinine clearance 50ml min, for full dose cyclo Creatinine clearance 10-50ml min for 75% of cyclo dose Creatinine clearance 10ml min for 50% of cyclo dose To calculate creatinine clearance: 140-age ; x wgt kg ; x1.23 for males 1.04 for females serum creatinine BEAM conditioning regimen Consult local chemotherapy transplant protocols for details of drug administration ; Day 6 Days 5, -4, -3, -2 Days 5, -4, -3, -2 Day 1 Carmustine Cytarabine Etoposide Melphalan 300mg m2 200mg m2 200mg m 140mg m and flecainide

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Smaller litter sizes. Although the fertility of the rats treated with the low dose of Cetrorelix appeared lower than that in those treated with Lupron, the differences were not statistically significant. All of the live-born pups appeared normal. In only 1 case, in the Lupron-treated group, 2 pups of a total of 86 in the group ; were killed by the mother. The lower fertility in experiment 3 than in experiment 1 was a result of the lower sperm counts Figure 3 ; caused by the higher dose of radiation and the. DOSAGE Diluted in about 1 4 glass of water or juice. Adults and Teenagers over 15: 1520 drops five minutes before bedtime. May be repeated during the night. Children under 15: Use Serenite Jr. Formula. HOW LONG DOES A BOTTLE LAST? One bottle will last approximately one month. INGREDIENTS Avena sativa, Scuttelaria laterifolia, Passiflora incarnata, Coffea C30, Nux vomica C30 and flexeril.

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Verapamil belongs to the group of calcium channel antagonists used in the management of essential hypertension. It is mostly used in a conventional tablet form from a minimal dose of 40 mg to a maximal dose of 180 mg. In a slow release tablet, the doses are between 120 and 240 mg. About 80% of the orally applied dose undergoes a first pass effect, mainly in the liver [3]. The preparation of floating pellets has been used to get a more effective release and absorption of verapamil due to a higher solubility of the drug in the stomach [3]. Another attempt to avoid problems of verapamil solubility by oral administration is the use of organic acids to maintain the pH low and the addition of enteric materials to open pores in a matrix at the pH of the intestine [4]. However, the still low bioavailability associated with the oral route can be avoided by transdermal administration [5]. Verapamil has a short half-life and requires frequent dosing. The permeability characteristics of verapamil show poor skin permeation properties; the permeation is dependent on the drug concentration and pH of the donor solution [6].

Ublic and private payers frequently use provider claims databases to track health care expenditures, analyze patterns of medical resource utilization, and provide a basis for making plan-level treatment decisions eg., drug coverage, treatment guidelines or restrictions, and medication formulary status ; . While claims-based analyses are increasingly used by decision makers due to low costs, rapidity of results, and large sample size, the sole reliance on claims data for making plan-level decisions is associated with notable challenges, limitations, and potential biases. The most significant limitations of claims data arise from deficiencies in the information that is available. Claims data typically lack critical data such as illness severity, the indication for drug use, the criteria used in making the treatment choice, and outcomes.1-4 In complex disease areas such as cancer, claims data present additional challenges such as a lack of information about the chemotherapy regimen used, the instances of and reasons for changes in the chemotherapy dose or schedule, and the cancer stage.1, 2 Further, claims databases may be subject to inconsistency over time as a result of ongoing editing and changes in coding and may contain invalid data due to a lack of edit checks.3 Missing or incorrect data can introduce serious bias, producing inaccurate and misleading results. Medical record review or linking of claims and registry databases can resolve many of these discrepancies but is sometimes considered too costly and difficult.1, 2 Finally, all data derived from administrative databases contain a non-random patient sample, which may introduce unknown bias into an analysis.3, 4 Because of these limitations, clinical guidelines and other important medical decisions should ideally be based on validated, methodologically sound data sources such as clinical trial results, medical chart reviews, or prospective studies. To illustrate the challenges and limitations of claims-based analyses compared with more methodologically rigorous techniques, we selected the use of granulocyte colony-stimulating factor ie., filgrastim ; in oncology as a case study. Filgrastim is indicated to decrease the incidence of infection, as manifested by febrile neutropenia, in patients with nonmyeloid malignancies receiving myelosuppressive anticancer drugs associated with a significant incidence of severe neutropenia with fever.5 In a decision-analytic model, the high costs of febrile neutropenia favored prophylactic filgrastim use compared with no use or therapeutic use.6 In randomized controlled clinical trials, filgrastim was administered for a mean of 11 days, beginning 24 hours after chemotherapy to reach an absolute neutrophil count ANC ; of 10 109 L--the level consistent with current prescribing information in the chemotherapy-induced neutropenia CIN ; setting. In these trials, the costs of purchasing and administering filgrastim were partially or completely offset by a reduction in the costs of febrile neutropenia.7-9 This article reports filgrastim use patterns based on analyses of and flolan.

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REFERENCES 1. Sheridan WP, Begley CG, Juttner CA, Szer J, To LB, Maher D, McGrath KM, Morstyn G , Fox R M : Effect of peripheral-blood progenitor cells mobilised by filgrastim G-CSF ; on platelet recovery after high-dose chemotherapy. Lancet 339540, 1992 2. Chao NJ, Schriber JR, Grimes K, Long GD, Negrin RS, Raimondi CM, Homing SJ, Brown SL, Miller L, Blume KG: Granulocyte colony-stimulating factor "mobilized" peripheral blood progenitor cells accelerate granulocyte and platelet recovery after high-dose chemotherapy. Blood 81: 2031. 1993 Bensinger W, Singer J, Appelbaum F, Lilleby K, Longin K, Rowley S , Clarke E, Clift R, Hansen J, Shields T, Storb R, Weaver C, Weiden P, Buckner CD: Autologous transplantation with peripheral blood mononuclear cells collected after administration of recombinant granulocyte stimulating factor. Blood 81: 3158, 1993 Peters WP, Rosner G, Ross M, Vredenburgh J, Meisenberg M, Gilbert C, Kurtzberg J: Comparative effects of granulocyte-macrophage colony-stimulating factor GM-CSF ; and granulocyte colonystimulating factor G-CSF ; on priming peripheral blood progenitor cells for use with autologous bone marrow after high-dose chemotherapy. Blood 81: 1709, 1993 Henon PhR, Liang H, Beck-Wirth G , Eisenmann JC, Lepers M, Wunder E, Kandel G: Comparison of hematopoietic and immune recovery after autologous bone marrow or blood stem cell transplants. Bone Marrow Transplant 9: 285, 1992 Roberts MM, To LB, Gillis D, Mundy J, Rawling C, Ng K ADDITIONAL INFORMATION Brain Donation Program "Give the Gift of Knowledge" Brain donation allows valuable information to be obtained regarding normal aging and diseases that affect memory. This information may be useful towards the goal of developing more effective treatment strategies and flu.

Promoters Zabaleta et al., 1998; Satoh et al., 2002; Thirkettle-Watts et al., 2003 ; or to analyze cell-specific expression Smart et al., 1994; Ribichich et al., 2001 ; . To characterize the regulation of nuclear genes encoding mitochondrial proteins and clarify further the roles of multiple SDH2 genes in complex II biogenesis, our group has undertaken a detailed analysis of SDH2 expression. In this study we analyze the expression of SDH2 transcripts by in situ hybridization, with emphasis in the process of flower development. Furthermore, transgenic Arabidopsis plants carrying progressive deletions of SDH2 promoters fused to the b-glucuronidase GUS ; reporter gene allowed us to define promoter regions involved in tissue-specific expression.
Br j haematol 1996, 94: 89-9 heil g, hoelzer d, sanz ma, et al: a randomized, double-blind, placebo-controlled, phase iii study of filgrastim in remission induction and consolidation therapy for adults with de novo acute myeloid leukemia and flucytosine. 1. Keating MJ, O'Brien S, Lerner S, et al: Long-term follow-up of patients with chronic lymphocytic leukemia CLL ; receiving fludarabine regimens as initial therapy. Blood 92: 1165-1171, 1998 Rai KB, Peterson B, Elias L, et al: A randomized comparison of fludarabine and chlorambucil for patients with previously untreated chronic lymphocytic leukemia: A CALBG, SWOG, CTG NCI-C and ECOG Intergroup study. Blood 88: 141a, 1997 suppl 1, abstr 40 ; 3. Tallman MS, Hakimian D: Purine nucleoside analogs: Emerging roles in indolent lymphoproliferative disorders. Blood 86: 2463-2474, 1995 The French Cooperative Group on CLL, Johnson S, Smith AG, et al: Multicentre prospective randomized trial of fludarabine versus cyclophosphamide, doxorubicin, and prednisone CAP ; for treatment of advanced-stage chronic lymphocytic leukemia. Lancet 347: 14321438, 1996 Fenchel K, Bergmann L, Wijermans P, et al: Clinical experience with fludarabine and its immunosuppressive effects in pretreated chronic lymphocytic leukemias and low-grade lymphomas. Leuk Lymphoma 18: 445-492, 1995 Hiddemann W, Rottmann R, Wormann B, et al: Treatment of advanced chronic lymphocytic leukemia by fludarabine: Results of a clinical study. Ann Hematol 63: 1-4, 1991 Keating MJ, Kantarjian H, Talpaz M, et al: Fludarabine: A new agent with major activity against chronic lymphocytic leukemia. Blood 74: 19-25, 1989 O'Brien S, Kantarjian H, Beran M, et al: Results of fludarabine and prednisone therapy in 264 patients with chronic lymphocytic leukemia with multivariate analysis-derived prognostic model for response to treatment. Blood 82: 1695-1700, 1993 Koehl U, Li L, Nowak B, et al: Fludarabine and cyclophosphamide: Synergistic cytotoxicity associated with inhibition of interstrand cross-link removal. Proc Assoc Cancer Res 38: 2, 1997 abstr 10 ; 10. Bellosillo B, Villamor N, Colomer D, et al: In vitro evaluation of fludarabine in combination with cyclophosphamide and or mitoxantrone in B-cell chronic lymphocytic leukemia. Blood 94: 2836-2843, 1999 Cheson BD, Bennett JM, Grever M, et al: National Cancer InstituteSponsored Working Group guidelines for chronic lymphocytic leukemia: Revised guidelines for diagnosis and treatment. Blood 87: 4990-4997, 1996 Mantel N: Evaluation of survival data and two new rank order statistics arising to consideration. Cancer Chemotherapy Rep 50: 163170, 1966 Kaplan EL, Meier P: Non-parametric estimation from incomplete observations. J Stat Assoc 53: 457-481, 1958 Larfars G, Uden-Blohme A-M, Samuelsson J: Fludarabine, as well as 2-chlorodeoxyadenosine, can induce eosinophilia during treatment of lymphoid malignancies. Br J Haematol 94: 709-712, 1996 Robak T, Blasinska-Morawiec M, Krykowski E, et al: 2-Chlorodeoxyadenosine cladribine ; -related eosinophilia in patients with lymphoproliferative diseases. Eur J Haematol 59: 216-220, 1997 Weiss M, Spiess T, Berman E, et al: Concomitant administration of chlorambucil limits dose intensity of fludarabine in previously treated patients with chronic lymphocytic leukemia. Leukemia 8: 12901293, 1994 Elias L, Stock-Novack D, Head D, et al: A phase I trial of combination fludarabine monophosphate and chlorambucil in chronic lymphocytic leukemia: A Southwest Oncology Group study. Leukemia 7: 361-365, 1993 Flinn I, Byrd J, Morrison C, et al: Fludarabine and cyclophosphamide with filgrastim support in patients with previously untreated indolent lymphoid malignancies. Blood 96: 71-75, 2000 Hallek M, Wilhelm M, Emerich B, et al: Fludarabine plus cyclophosphamide FC ; and dose-intensified chlorambucil DIC ; for the treatment of chronic lymphocytic leukemia CLL ; : Results of two phase II studies CLL2-protocol ; of the German CLL Study Group GCLLSG ; . Blood 11: 313a, 1999 abstr 1402 and filgrastim.

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43 product general subject matter expiration epoetin alfa — process of making erythropoietin 8 15 2012 — product claims to erythropoietin 8 20 2013 — pharmaceutical compositions of erythropoietin 8 20 2013 — cells that make certain levels of erythropoietin 5 26 2015 darbepoetin alfa europe 1 ; — glycosylation analogs of erythropoietin proteins 10 12 2010 — glycosylation analogs of erythropoietin proteins 8 16 2014 filgrastim — g-csf polypeptides 12 3 2013 — methods of treatment using g-csf polypeptides 12 10 2013 pegfilgrastim — pegylated g-csf 10 20 2015 europe 1 ; — pegylated g-csf 2 8 2015 etanercept — methods of treating tnf — dependent disease 9 5 2009 — tnfr proteins and pharmaceutical compositions 9 5 2009 — tnfr dna vectors, cells and processes for making proteins 10 23 2012 ; in some cases these european patents may also be entitled to supplemental protection in one or more countries in europe and the length of any such extension will vary country by country and fludarabine.
1.4 "API" means the active pharmaceutical ingredient contained in the Sanvar Product, being vapreotide acetate, manufactured in accordance with the Specifications. 1.5 "Approvable Letter" means the approvable letter dated December 21, 2004 from the FDA to Debiovision in respect of the Existing NDA. 1.6 "Approved Manufacturer" means, as the case may be, either the current and or future FDA approved manufacturer for the API or the current and or future FDA approved manufacturer for the Sanvar Product, appointed and approved in accordance with Section 9. 1.7 "Business Day" means a day from 9: 00 to local time on a day other than a Saturday, Sunday or bank or other public holiday in the United States of America or Canada. 1.8 "Claimed" means in respect of any product, material or formulation that it is within a Valid Claim of any Patent Rights. 1.9 "Clinical Development" means any Clinical Study managed by Salix which may be necessary or required by the Regulatory Authority of the Territory in order to obtain or maintain Regulatory Approval of a Product for any Indication in the Territory. 1.10 "Clinical Study" means any clinical study carried out by or on behalf of Salix during the Initial Term i ; relating to the Sanvar Product post NDA Approval Date, including, without limitation, any study carried out in order to obtain a label extension to the Sanvar Product; ii ; in connection with any NDA for any Product in the Territory; or iii ; as part of any Lifecycle Management Plan for any Product. 1.11 "Combination Product" means any Product which comprises the Technology in combination with one or more other active ingredients, identified by either Party during the Initial Term. 1.12 "Commercialization", "Commercializing", or "Commercialize" means all activities relating to the advertising, promotion and other marketing, pricing and reimbursement, Detailing, distribution, storage, handling, offering for sale and selling, customer service and support of a Product. 1.13 "Commercialization Plan" means the plan to be submitted to Debiovision and agreed by the JSC and all related annual updated versions, under which Salix commits to Commercialize the Sanvar Product with planned minimum resource requirements for the Territory and subsequently, commits to commercialise any New Product or Combination Product. The Commercialization Plan shall include i ; Salix Commitment including minimum promotional and marketing budget and expenditures ii ; anticipated Launch dates; iii ; anticipated market share; and iv ; Sales Forecasts. 1.14 "Competing Product" means any human pharmaceutical product that is not a Prohibited Product, that i ; contains the same active ingredient s ; as that those contained in the Sanvar Product; or ii ; that contains or is a vasoactive agent; and is a substitute for or directly competitive with the Sanvar Product in the Territory, and for the avoidance of doubt, a Generic other than a Salix Generic ; may be a Competing Product. 4.

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G-CSF AFTER CONSOLIDATION IN AML 6. Ravindranath Y, Yeager AM, Chang MN, et al: Autologous bone marrow transplantation versus intensive consolidation chemotherapy for acute myeloid leukemia in childhood. N Engl J Med 334: 14281434, 1996 Harousseau JL, Cahn JY, Pignon B, et al: Comparison of autologous bone marrow transplantation and intensive chemotherapy as postremission therapy in adult acute myeloid leukemia. Blood 90: 29782986, 1997 Cassileth PA, Harrington DP, Appelbaum FR, et al: Chemotherapy compared with autologous or allogeneic bone marrow transplantation in the management of acute myeloid leukemia in first remission. N Engl J Med 339: 1649-1656, 1998 Schiffer CA: Hematopoietic growth factors as adjuncts to the treatment of acute myeloid leukemia. Blood 88: 3675-3685, 1996 Terpstra W, Lowenberg B: Application of myeloid growth factors in the treatment of acute myeloid leukemia. Leukemia 11: 315327, 1997 Rowe JM, Liesveld JL: Hematopoietic growth factors in acute leukemia. Leukemia 11: 328-341, 1997 Harousseau JL: The role of colony stimulating factors in the treatment of acute leukemia. Biodrugs 6: 448-460, 1997 Bennett JM, Catovsky D, Daniel MT, et al: Proposed revised criteria for the classification of acute myeloid leukemia: A report of the French-American-British Cooperative group. Ann Intern Med 103: 626629, 1985 Dastugue N, Payen C, Lafage-Pochitaloff M, et al: Prognostic significance of Karyotype in de novo adult myeloid leukemia. Leukemia 9: 1491-1498, 1995 Grimwade D, Walker H, Oliver F, et al: The importance of diagnostic cytogenetics on outcome in AML: Analysis of 1612 patients entered into the MRC AML 10 trial. Blood 92: 2322-2333, 1998 Solary E, Witz B, Caillot D, et al: Combination of quinine as a potential reversing agent with mitoxantrone and cytarabine for the treatment of acute leukemias: A randomized multicenter study. Blood 88: 1198-1205, 1996 Dombret H, Chastang C, Fenaux P, et al: A controlled study of recombinant human granulocyte colony stimulating factor in elderly patients after treatment for acute myelogenous leukemia. N Engl J Med 232: 1678-1683, 1995 Rowe JM, Andersen JW, Mazza JJ, et al: A randomized placebo-controlled phase III study of granulocyte-macrophage colonystimulating factor in adult patients 55 to 70 years of age ; with acute myelogenous leukemia: A study of the Eastern Cooperative Oncology Group. Blood 86: 457-462, 1995 Stone RM, Berg DT, George SL, et al: Granulocyte-macrophage colony-stimulating factor after initial chemotherapy for elderly patients with primary acute myelogenous leukemia. N Engl J Med 332: 16711677, 1995 Heil G, Hoelzer D, Sanz MA, et al: A randomized double blind placebo-controlled phase III study of filgrastim in remission induction and flumist.

Acknowledgments From Kaiser Permanente Northern California, Andrew Bertagnolli, PhD, assisted with the literature review; and Marilee Donovan, PhD, Manager Clinical Nurse Specialist, Pain Management Clinic, provided editorial assistance. 6. References 1. Turk DC. Treatment of chronic pain: clinical outcomes, cost effectiveness and cost benefits. Drug Benefit Trends 2001; 13: 36-8. Cherny NI, Portenoy RK. The management of cancer pain. CA Cancer J Clin 1994 SepOct; 44 5 ; : 263-303. 3. Mosen D. KP Health Impact Assessment HIP ; : results from a pilot project to collect quality of life information using Internet and interactive voice recognition IVR ; technologies [Powerpoint presentation on the Intranet]. [accessed 2004 Nov 18]. Available from: : kpnet.kp qrrm nqc04 presentations 2. 4. Eimer BN, Freeman A. Pain management psychotherapy: a practical guide. New York: John Wiley & Sons, Inc; 1998. 5. Kaiser Permanente. Care Manage and flax.

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