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The author wishes to thank his colleague Ellen McKenney Frankfurt Main ; for her dedication and patience in proofreading and correcting the English version of this paper. 2 All quotations in this paper are translations from the Russian original; the translations were done by the author. Transliteration of text from the Cyrillic to the Latin alphabet is based on the International Scholarly System "Scientific Transliteration" ; . Such general rule notwithstanding, names of internationally known public figures are romanised according to common practise in English e.g. "Khrushchev" instead of "Hruscev" ; . Russia has a very complex history of reading literacy and of media to support learning to read. This complexity is partly reflected in terminology. Whereas the term bukvar' is usually reserved to denominate school textbooks for first-graders, azbuka can designate basically three types of books: a ; an ABC-book which presents the letters in alphabetical order according to the Russian alphabet: from a to ja ; , usually one letter per page, accompanied by one ore more pictures; b ; an educational aid to support learning to read at home, with a more or less elaborated didactical design; c ; a textbook for reading instruction in educational establishments kindergartens and schools ; . The term "primer" is used in this paper to designate both bukvary and azbuki as far as they are designed to be used as textbooks in educational establishments and triac
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Figure 4. Top: Family pedigree of proband III-3 ; . Circles and squares represent females and males, respectively. Open symbols indicate unaffected family members, the dashed symbol III-2 ; indicates an obligate carrier of mutation, and closed symbols represent affected individuals with pulmonary hypertension. Deceased family members are marked by diagonal lines. Underneath each individual are their haplotypes with the maternal haplotype first see table below * indicates the haplotype carrying the del44C bone morphogenetic protein receptor II BMPR2 ; mutation. For those family members for whom no DNA samples were available for analysis, the haplotypes have been inferred see parentheses ; on the basis of Mendelian inheritance. Of note, the assignment of haplotypes to the grandparents I-1 and I-2 ; was random as either of them may have passed on the mutant haplotype. Middle: Digestion patterns of amplicons generated by polymerase chain reaction of DNAs from family members after digestion with Nco I restriction endonuclease and electrophoresis on 2% agarose gel stained with ethidium bromide. Patterns correspond to individuals in the above pedigree; the proband's mother II-5 ; did not have a sample for analysis and thus the lane beneath her is empty. In the heterozygous state, because the mutation del44C destroys an Nco I restriction endonuclease site, it is detected by the undigested amplicon at 602 base pairs bp ; . DNAs with 602 and 467 bp fragments are therefore heterozygous for the mutant and normal allele. The bands at the far left of the gel are a standard DNA ladder Low Biomarker, Bioventures, Murfreesboro, TN ; with their sizes listed to the left. Bottom: Table listing the haplotypes in this family for four polymorphic dinucleotide loci flanking the BMPR2 locus D2S311D2S116BMPR2D2S307D2S369 and torsemide.
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1. Arnett RH III, Blank LA, Brown AP, et al. National health expenditures, 1988: Office of National Cost Estimates. Health Care Financ Rev. 1990; 11: 1-41. Hanlon JT, Schmader KE, Ruby CM, et al. Suboptimal prescribing in older inpatients and outpatients. J Geriatr Soc. 2001; 49: 200-209. Brawn LA, Castleden CM. Adverse drug reactions: an overview of special considerations in the management of the elderly patient. Drug Saf. 1990; 5: 421435. Carbonin P, Pahor M, Bernabei R, et al. Is age an independent risk factor of adverse drug reactions in hospitalized medical patients? J Geriatr Soc. 1991; 39: 1093-1099. Gurwitz JH, Avorn J. The ambiguous relation between aging and adverse drug reactions. Ann Intern Med. 1991; 114: 956-966. Stewart RB, Cooper JW. Polypharmacy in the aged: practical solutions. Drugs Aging. 1994; 4: 449-461. Huskamp HA, Rosenthal MB, Frank RG, et al. The Medicare prescription drug benefit: how will the game be played? Health Aff Millwood ; . 2000; 19: 8-23. Goff VV. Pharmacy benefit managers: a model for Medicare? Issue Brief Natl Health Policy Forum. 2001; 765: 1-12. Beers MH, Ouslander JG, Rollingher I, et al. Explicit criteria for determining inappropriate medication use in nursing homes. Arch Intern Med. 1991; 151: 18251832. Beers MH, Ouslander JG, Fingold SF, et al. Inappropriate medication prescribing in skilled-nursing facilities. Ann Intern Med. 1992; 117: 684-689.
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26. Howes LG, Hodsman GP, Rowe PR et al. Comparative effects of angiotensin converting enzyme inhibition perindopril ; or diuretic therapy on cardiac hypertrophy and sympathetic activity following myocardial infarction in rats. Cardiovasc Drugs Ther. 1991; 5: 147-152. Kohzuki M, Kanazawa M, Yoshida K et al. Cardiomegaly and vasoactive hormones in rats with chronic myocardial infarction: long-term effects of chlorothiazide. Clin Sci Colch ; . 1996; 90: 31-36. Seeland U, Kouchi I, Zolk O et al. Effect of ramipril and furosemide treatment on interstitial remodeling in post-infarction heart failure rat hearts. J Mol Cell Cardiol. 2002; 34: 151-163. Hodsman GP, Sumithran E, Harrison RW et al. Cardiac hypertrophy and salt status in chronic myocardial infarction in the rat: effects of enalapril versus salt restriction. J Cardiovasc Pharmacol. 1988; 12: 467-472. Hodsman GP, Harrison RW, Sumithran E et al. Atrial natriuretic peptide and total exchangeable body sodium: relationships in rats with chronic myocardial infarction. Clin Sci Lond ; . 1988; 74: 659-663. Dormans TP, Pickkers P, Russel FG et al. Vascular effects of loop diuretics. Cardiovasc Res. 1996; 32: 988997. Pickkers P, Hughes AD, Russel FG et al. Thiazide-induced vasodilation in humans is mediated by potassium channel activation. Hypertension. 1998; 32: 1071-1076. Pickkers P, Dormans TP, Russel FG et al. Direct vascular effects of furosemide in humans. Circulation. 1997; 96: 1847-1852. Zhu Z, Zhu S, Liu D et al. Thiazide-like diuretics attenuate agonist-induced vasoconstriction by calcium desensitization linked to rho kinase. Hypertension. 2005; 45: 233-239. Cosin J, Diez J. Torasemide in chronic heart failure: results of the TORIC study. Eur J Heart Fail. 2002; 4: 507-513. Lopez B, Querejeta R, Gonzalez A et al. Effects of loop diuretics on myocardial fibrosis and collagen type I turnover in chronic heart failure. J Coll Cardiol. 2004; 43: 2028-2035. Goodfriend TL, Ball DL, Oelkers W et al. Torsemide inhibits aldosterone secretion in vitro. Life Sci. 1998; 63: L45-L50. 38. Tsutamoto T, Sakai H, Wada A et al. Torasemide inhibits transcardiac extraction of aldosterone in patients with congestive heart failure. J Coll Cardiol. 2004; 44: 2252-2253. Pitt B, Remme W, Zannad F et al. Eplerenone, a selective aldosterone blocker, in patients with left ventricular dysfunction after myocardial infarction. N Engl J Med. 2003; 348: 1309-1321. Pitt B, Zannad F, Remme WJ et al. The effect of spironolactone on morbidity and mortality in patients with severe heart failure. Randomized Aldactone Evaluation Study Investigators. N Engl J Med. 1999; 341: 709-717. Ferrario CM, Chappell MC, Tallant EA et al. Counterregulatory actions of angiotensin- 1-7 ; . Hypertension. 1997; 30: 535-541. Loot AE, Roks AJ, Henning RH et al. Angiotensin- 1-7 ; attenuates the development of heart failure after myocardial infarction in rats. Circulation. 2002; 105: 1548-1550. Kocks MJ, Titia Lely A, Boomsma F et al. Sodium status and angiotensin-converting enzyme inhibition: effects on plasma angiotensin- 1-7 ; in healthy man. J Hypertens. 2005; 23: 597-602. Gross V, Obst M, Luft FC. Insights into angiotensin II receptor function through AT2 receptor knockout mice. Acta Physiol Scand. 2004; 181: 487-494. Siragy HM, Xue C, Abadir P et al. Angiotensin subtype-2 receptors inhibit renin biosynthesis and angiotensin II formation. Hypertension. 2005; 45: 133-137. Gonzalez M, Lobos L, Castillo F et al. High-salt diet inhibits expression of angiotensin type 2 receptor in resistance arteries. Hypertension. 2005; 45: 853-859 and trihexyphenidyl.
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